RESUMO
Quasiholes in certain fractional quantum Hall states are promising candidates for the experimental realization of non-Abelian anyons. They are assumed to be localized excitations, and to display non-Abelian statistics when sufficiently separated, but these properties have not been explicitly demonstrated except for the Moore-Read state. In this work, we apply the newly developed matrix product state technique to examine these exotic excitations. For the Moore-Read and the Z_{3} Read-Rezayi states, we estimate the quasihole radii, and determine the correlation lengths associated with the exponential convergence of the braiding statistics. We provide the first microscopic verification for the Fibonacci nature of the Z_{3} Read-Rezayi quasiholes. We also present evidence for the failure of plasma screening in the nonunitary Gaffnian wave function.
RESUMO
We introduce a Bloch-like basis in a C-component lowest Landau level fractional quantum Hall (FQH) effect, which entangles the real and internal degrees of freedom and preserves an N(x)×N(y) full lattice translational symmetry. We implement the Haldane pseudopotential Hamiltonians in this new basis. Their ground states are the model FQH wave functions, and our Bloch basis allows for a mutatis mutandis transcription of these model wave functions to the fractional Chern insulator of arbitrary Chern number C, obtaining wave functions different from all previous proposals. For C>1, our wave functions are related to color-dependent magnetic-flux inserted versions of Halperin and non-Abelian color-singlet states. We then provide large-size numerical results for both the C = 1 and C = 3 cases. This new approach leads to improved overlaps compared to previous proposals. We also discuss the adiabatic continuation from the fractional Chern insulator to the FQH in our Bloch basis, both from the energy and the entanglement spectrum perspectives.
RESUMO
Attribution of biological robustness to the specific structural properties of a regulatory network is an important yet unsolved problem in systems biology. It is widely believed that the topological characteristics of a biological control network largely determine its dynamic behavior, yet the actual mechanism is still poorly understood. Here, we define a novel structural feature of biological networks, termed 'regulation entropy', to quantitatively assess the influence of network topology on the robustness of the systems. Using the cell-cycle control networks of the budding yeast (Saccharomyces cerevisiae) and the fission yeast (Schizosaccharomyces pombe) as examples, we first demonstrate the correlation of this quantity with the dynamic stability of biological control networks, and then we establish a significant association between this quantity and the structural stability of the networks. And we further substantiate the generality of this approach with a broad spectrum of biological and random networks. We conclude that the regulation entropy is an effective order parameter in evaluating the robustness of biological control networks. Our work suggests a novel connection between the topological feature and the dynamic property of biological regulatory networks.
Assuntos
Modelos Biológicos , Biologia de Sistemas/métodos , Algoritmos , Ciclo Celular/fisiologia , Saccharomycetales/fisiologia , Schizosaccharomyces/fisiologia , Transdução de Sinais , TermodinâmicaRESUMO
In a ferroin-catalyzed Belousov-Zhabotinsky (BZ) reaction-diffusion system with reagent concentration gradients, we observed in the experiment a type of spirals with local waves forming groups. Here, we propose an interpretation of the wave grouping phenomenon. The wave grouping mechanism can be well explained in terms of the cooperation of the excitability gradient and the Doppler effect induced by spiral tip's meandering. In the simulation based on three-dimensional reaction-diffusion system using Oregonator model, spiral patterns analogous to the experiment observation are well reproduced when the parameter gradient in the z direction is introduced.
RESUMO
One of the major objectives in systems biology is to understand the relation between the topological structures and the dynamics of biological regulatory networks. In this context, various mathematical tools have been developed to deduct structures of regulatory networks from microarray expression data. In general, from a single data set, one cannot deduct the whole network structure; additional expression data are usually needed. Thus how to design a microarray expression experiment in order to get the most information is a practical problem in systems biology. Here we propose three methods, namely, maximum distance method, trajectory entropy method, and sampling method, to derive the optimal initial conditions for experiments. The performance of these methods is tested and evaluated in three well-known regulatory networks (budding yeast cell cycle, fission yeast cell cycle, and E. coli. SOS network). Based on the evaluation, we propose an efficient strategy for the design of microarray expression experiments.