Detalhe da pesquisa
1.
Best practices in assessing cardiac protein O-GlcNAcylation by immunoblot.
Am J Physiol Heart Circ Physiol
; 325(4): H601-H616, 2023 10 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-37539459
2.
Acute increases in O-GlcNAc indirectly impair mitochondrial bioenergetics through dysregulation of LonP1-mediated mitochondrial protein complex turnover.
Am J Physiol Cell Physiol
; 316(6): C862-C875, 2019 06 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-30865517
3.
Novel role of the ER/SR Ca2+ sensor STIM1 in the regulation of cardiac metabolism.
Am J Physiol Heart Circ Physiol
; 316(5): H1014-H1026, 2019 05 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-30575437
4.
Stromal interaction molecule 1 is essential for normal cardiac homeostasis through modulation of ER and mitochondrial function.
Am J Physiol Heart Circ Physiol
; 306(8): H1231-9, 2014 Apr 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-24585777
5.
High-fat, low-carbohydrate diet promotes arrhythmic death and increases myocardial ischemia-reperfusion injury in rats.
Am J Physiol Heart Circ Physiol
; 307(4): H598-608, 2014 Aug 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-24929857
6.
Modification of STIM1 by O-linked N-acetylglucosamine (O-GlcNAc) attenuates store-operated calcium entry in neonatal cardiomyocytes.
J Biol Chem
; 287(46): 39094-106, 2012 Nov 09.
Artigo
em Inglês
| MEDLINE | ID: mdl-22992728
7.
Glucose deprivation-induced increase in protein O-GlcNAcylation in cardiomyocytes is calcium-dependent.
J Biol Chem
; 287(41): 34419-31, 2012 Oct 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-22908225
8.
Sustained Increases in Cardiomyocyte Protein O-Linked ß-N-Acetylglucosamine Levels Lead to Cardiac Hypertrophy and Reduced Mitochondrial Function Without Systolic Contractile Impairment.
J Am Heart Assoc
; 12(19): e029898, 2023 10 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-37750556
9.
O-GlcNAcylation, novel post-translational modification linking myocardial metabolism and cardiomyocyte circadian clock.
J Biol Chem
; 286(52): 44606-19, 2011 Dec 30.
Artigo
em Inglês
| MEDLINE | ID: mdl-22069332
10.
Aging influences cardiac mitochondrial gene expression and cardiovascular function following hemorrhage injury.
Mol Med
; 17(5-6): 542-9, 2011.
Artigo
em Inglês
| MEDLINE | ID: mdl-21193900
11.
The Identification of a Novel Calcium-Dependent Link Between NAD+ and Glucose Deprivation-Induced Increases in Protein O-GlcNAcylation and ER Stress.
Front Mol Biosci
; 8: 780865, 2021.
Artigo
em Inglês
| MEDLINE | ID: mdl-34950703
12.
Importance of the bioenergetic reserve capacity in response to cardiomyocyte stress induced by 4-hydroxynonenal.
Biochem J
; 424(1): 99-107, 2009 Oct 23.
Artigo
em Inglês
| MEDLINE | ID: mdl-19740075
13.
The protective effects of PUGNAc on cardiac function after trauma-hemorrhage are mediated via increased protein O-GlcNAc levels.
Shock
; 27(4): 402-8, 2007 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-17414423
14.
Glucosamine administration during resuscitation improves organ function after trauma hemorrhage.
Shock
; 25(6): 600-7, 2006 Jun.
Artigo
em Inglês
| MEDLINE | ID: mdl-16721268
15.
Assessing bioenergetic function in response to oxidative stress by metabolic profiling.
Free Radic Biol Med
; 51(9): 1621-35, 2011 Nov 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-21872656
16.
Glucosamine improves cardiac function following trauma-hemorrhage by increased protein O-GlcNAcylation and attenuation of NF-{kappa}B signaling.
Am J Physiol Heart Circ Physiol
; 296(2): H515-23, 2009 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-19098112
17.
Adult rat cardiomyocytes exhibit capacitative calcium entry.
Am J Physiol Heart Circ Physiol
; 286(3): H1124-32, 2004 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-14630640