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Reciprocal interactions between mitral valve endothelial and interstitial cells reduce endothelial-to-mesenchymal transition and myofibroblastic activation.
Shapero, Kayle; Wylie-Sears, Jill; Levine, Robert A; Mayer, John E; Bischoff, Joyce.
Afiliação
  • Shapero K; Vascular Biology Program, Department of Surgery, Boston Children's Hospital, Harvard Medical School, USA; Biomedical Engineering Department, Boston University, Boston, MA, USA; Department of Cardiothoracic Surgery, Boston Children's Hospital, Harvard Medical School, USA.
  • Wylie-Sears J; Vascular Biology Program, Department of Surgery, Boston Children's Hospital, Harvard Medical School, USA.
  • Levine RA; Cardiac Ultrasound Laboratory, Massachusetts General Hospital, Harvard Medical School, Boston, MA 02114, USA.
  • Mayer JE; Department of Cardiothoracic Surgery, Boston Children's Hospital, Harvard Medical School, USA.
  • Bischoff J; Vascular Biology Program, Department of Surgery, Boston Children's Hospital, Harvard Medical School, USA. Electronic address: joyce.bischoff@childrens.harvard.edu.
J Mol Cell Cardiol ; 80: 175-85, 2015 Mar.
Article em En | MEDLINE | ID: mdl-25633835
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Comunicação Celular / Células Endoteliais / Transdiferenciação Celular / Miofibroblastos Idioma: En Ano de publicação: 2015 Tipo de documento: Article País de afiliação: Estados Unidos
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Comunicação Celular / Células Endoteliais / Transdiferenciação Celular / Miofibroblastos Idioma: En Ano de publicação: 2015 Tipo de documento: Article País de afiliação: Estados Unidos