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Hyperaldosteronism after decreased renal K+ excretion in KCNMB2 knockout mice.
Larsen, Casper K; Jensen, Iben S; Sorensen, Mads V; de Bruijn, Pauline I; Bleich, Markus; Praetorius, Helle A; Leipziger, Jens.
Afiliação
  • Larsen CK; Department of Biomedicine, Physiology, and Health, Aarhus University, Aarhus, Denmark;
  • Jensen IS; Department of Biomedicine, Physiology, and Health, Aarhus University, Aarhus, Denmark;
  • Sorensen MV; Department of Biomedicine, Physiology, and Health, Aarhus University, Aarhus, Denmark; Aarhus Institute for Advanced Studies, Aarhus University, Aarhus, Denmark; and.
  • de Bruijn PI; Department of Biomedicine, Physiology, and Health, Aarhus University, Aarhus, Denmark;
  • Bleich M; Institute of Physiology, Christian-Albrechts-University, Kiel, Germany.
  • Praetorius HA; Department of Biomedicine, Physiology, and Health, Aarhus University, Aarhus, Denmark;
  • Leipziger J; Department of Biomedicine, Physiology, and Health, Aarhus University, Aarhus, Denmark; leip@biomed.au.dk.
Am J Physiol Renal Physiol ; 310(10): F1035-46, 2016 05 15.
Article em En | MEDLINE | ID: mdl-26962098
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Potássio / Subunidades beta do Canal de Potássio Ativado por Cálcio de Condutância Alta / Hiperaldosteronismo / Rim Idioma: En Ano de publicação: 2016 Tipo de documento: Article
Texto completo
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XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Potássio / Subunidades beta do Canal de Potássio Ativado por Cálcio de Condutância Alta / Hiperaldosteronismo / Rim Idioma: En Ano de publicação: 2016 Tipo de documento: Article