Plasma shielding removes prior magnetization record from impacted rocks near Santa Fe, New Mexico.

The shock exposure of the Santa Fe's impact structure in New Mexico is evidenced by large human-size shatter cones. We discovered a new magnetic mechanism that allows a magnetic detection of plasma's presence during the impact processes. Rock fragments from the impactites were once magnetized by a geomagnetic field. Our novel approach, based on Neel's theory, revealed more than an order of magnitude lower magnetizations in the rocks that were exposed to the shockwave. Here we present a support for a newly proposed mechanism where the shock wave appearance can generate magnetic shielding that allow keeping the magnetic grains in a superparamagnetic-like state shortly after the shock's exposure, and leaves the individual magnetized grains in random orientations, significantly lowering the overall magnetic intensity. Our data not only clarify how an impact process allows for a reduction of magnetic paleointensity but also inspire a new direction of effort to study impact sites, using paleointensity reduction as a new impact proxy.

Analysis of dermal fibroblasts isolated from neonatal and child cleft lip and adult skin: Developmental implications on reconstructive surgery.

The nonsyndromic cleft is one of the most frequent congenital defects in humans. Clinical data demonstrated improved and almost scarless neonatal healing of reparative surgery. Based on our previous results on crosstalk between neonatal fibroblasts and adult keratinocytes, the present study focused on characterization of fibroblasts prepared from cleft lip tissue samples of neonates and older children, and compared them with samples isolated from normal adult skin (face and breast) and scars. Although subtle variances in expression profiles of children and neonates were observed, the two groups differed significantly from adult cells. Compared with adult cells, differences were observed in nestin and smooth muscle actin (SMA) expression at the protein and transcript level. Furthermore, fibroblast to myofibroblast differentiation drives effective wound healing and is largely regulated by the cytokine, transforming growth factor-ß1 (TGF-ß1). Dysregulation of the TGF-ß signalling pathway, including low expression of the TGF-ß receptor II, may contribute to reducing scarring in neonates. Fibroblasts of facial origin also exhibited age independent differences from the cells prepared from the breast, reflecting the origin of the facial cells from neural crest-based ectomesenchyme.