Detalhe da pesquisa
1.
SYSTEMI - systemic organ communication in STEMI: design and rationale of a cohort study of patients with ST-segment elevation myocardial infarction.
BMC Cardiovasc Disord
; 23(1): 232, 2023 05 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-37138228
2.
Regulation of titin-based cardiac stiffness by unfolded domain oxidation (UnDOx).
Proc Natl Acad Sci U S A
; 117(39): 24545-24556, 2020 09 29.
Artigo
em Inglês
| MEDLINE | ID: mdl-32929035
3.
Diabetes disturbs functional adaptation of the remote myocardium after ischemia/reperfusion.
J Mol Cell Cardiol
; 173: 47-60, 2022 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-36150524
4.
Life cycle assessment of animal-based foods and plant-based protein-rich alternatives: an environmental perspective.
J Sci Food Agric
; 102(12): 5098-5110, 2022 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-34231208
5.
Cardiac Hyaluronan Synthesis Is Critically Involved in the Cardiac Macrophage Response and Promotes Healing After Ischemia Reperfusion Injury.
Circ Res
; 124(10): 1433-1447, 2019 05 10.
Artigo
em Inglês
| MEDLINE | ID: mdl-30916618
6.
Diabetes-Induced Cardiomyocyte Passive Stiffening Is Caused by Impaired Insulin-Dependent Titin Modification and Can Be Modulated by Neuregulin-1.
Circ Res
; 123(3): 342-355, 2018 07 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-29760016
7.
Smyd2 controls cytoplasmic lysine methylation of Hsp90 and myofilament organization.
Genes Dev
; 26(2): 114-9, 2012 Jan 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-22241783
8.
The origin of myocardial passive stiffness: more than the sum of its parts?
Pflugers Arch
; 476(5): 715-716, 2024 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-38418696
9.
Impaired Ca2+ cycling of nonischemic myocytes contributes to sarcomere dysfunction early after myocardial infarction.
J Mol Cell Cardiol
; 119: 28-39, 2018 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-29674140
10.
Myofilament Remodeling and Function Is More Impaired in Peripartum Cardiomyopathy Compared with Dilated Cardiomyopathy and Ischemic Heart Disease.
Am J Pathol
; 187(12): 2645-2658, 2017 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-28935576
11.
Titin-Based Cardiac Myocyte Stiffening Contributes to Early Adaptive Ventricular Remodeling After Myocardial Infarction.
Circ Res
; 119(9): 1017-1029, 2016 Oct 14.
Artigo
em Inglês
| MEDLINE | ID: mdl-27650557
12.
Genotype-specific pathogenic effects in human dilated cardiomyopathy.
J Physiol
; 595(14): 4677-4693, 2017 07 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-28436080
13.
Remote ischemic preconditioning and beyond-are we getting closer to the secret of cardioprotection?
Pflugers Arch
; 471(11-12): 1369-1370, 2019 12.
Artigo
em Inglês
| MEDLINE | ID: mdl-31631253
14.
Titin: central player of hypertrophic signaling and sarcomeric protein quality control.
Biol Chem
; 395(11): 1341-52, 2014 Nov 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-25205716
15.
The challenges of research data management in cardiovascular science: a DGK and DZHK position paper-executive summary.
Clin Res Cardiol
; 113(5): 672-679, 2024 May.
Artigo
em Inglês
| MEDLINE | ID: mdl-37847314
16.
Corrigendum to 'Impaired Ca2+ cycling of nonischemic myocytes contributes to sarcomere dysfunction early after myocardial infarction' [J. Mol. Cell. Cardiol. 119 (2018) 28-39].
J Mol Cell Cardiol
; 121: 304, 2018 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-29907408
17.
Molecular and cellular evidence for the impact of a hypertrophic cardiomyopathy-associated RAF1 variant on the structure and function of contractile machinery in bioartificial cardiac tissues.
Commun Biol
; 6(1): 657, 2023 06 21.
Artigo
em Inglês
| MEDLINE | ID: mdl-37344639
18.
The giant protein titin: a regulatory node that integrates myocyte signaling pathways.
J Biol Chem
; 286(12): 9905-12, 2011 Mar 25.
Artigo
em Inglês
| MEDLINE | ID: mdl-21257761
19.
Sildenafil and B-type natriuretic peptide acutely phosphorylate titin and improve diastolic distensibility in vivo.
Circulation
; 124(25): 2882-91, 2011 Dec 20.
Artigo
em Inglês
| MEDLINE | ID: mdl-22144574
20.
Editorial on EMC 2017 special issue.
J Muscle Res Cell Motil
; 38(3-4): 271-273, 2017 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-29170862