Detalhe da pesquisa
1.
Deficiency of IKKß in neurons ameliorates Alzheimer's disease pathology in APP- and tau-transgenic mice.
FASEB J
; 37(2): e22778, 2023 02.
Artigo
em Inglês
| MEDLINE | ID: mdl-36688823
2.
P38α-MAPK phosphorylates Snapin and reduces Snapin-mediated BACE1 transportation in APP-transgenic mice.
FASEB J
; 35(7): e21691, 2021 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-34118085
3.
Neuronal deficiency of p38α-MAPK ameliorates symptoms and pathology of APP or Tau-transgenic Alzheimer's mouse models.
FASEB J
; 34(7): 9628-9649, 2020 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-32475008
4.
Deficiency of Neuronal p38α MAPK Attenuates Amyloid Pathology in Alzheimer Disease Mouse and Cell Models through Facilitating Lysosomal Degradation of BACE1.
J Biol Chem
; 291(5): 2067-79, 2016 Jan 29.
Artigo
em Inglês
| MEDLINE | ID: mdl-26663083
5.
Deficiency of IκB Kinase ß in Myeloid Cells Reduces Severity of Experimental Autoimmune Encephalomyelitis.
Am J Pathol
; 186(5): 1245-57, 2016 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-26968344
6.
p38α-MAPK-deficient myeloid cells ameliorate symptoms and pathology of APP-transgenic Alzheimer's disease mice.
Aging Cell
; 21(8): e13679, 2022 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-35909315