GADD45-induced cell cycle G2-M arrest associates with altered subcellular distribution of cyclin B1 and is independent of p38 kinase activity.

Jin, Shunqian; Tong, Tong; Fan, Wenhong; Fan, Feiyue; Antinore, Michael J; Zhu, Xiaocheng; Mazzacurati, Lucia; Li, Xianxing; Petrik, Kimberly L; Rajasekaran, Baskaran; Wu, Min; Zhan, Qimin

Jin, Shunqian; Tong, Tong; Fan, Wenhong; Fan, Feiyue; Antinore, Michael J; Zhu, Xiaocheng; Mazzacurati, Lucia; Li, Xianxing; Petrik, Kimberly L; Rajasekaran, Baskaran; Wu, Min; Zhan, Qimin.

Affiliation

Jin S; Department of Radiation Oncology, Cancer Institute, University of Pittsburgh School of Medicine, Pittsburgh, Pennsylvania, PA 15213, USA.

Oncogene ; 21(57): 8696-704, 2002 Dec 12.

Article in En | MEDLINE | ID: mdl-12483522

Subject(s)

Cyclin B/metabolism; G2 Phase/genetics; Mitogen-Activated Protein Kinases/metabolism; Mitosis/genetics; Proteins/genetics; Subcellular Fractions/metabolism; Checkpoint Kinase 1; Cyclin B1; HeLa Cells; Humans; Intracellular Signaling Peptides and Proteins; Phosphorylation; Protein Kinases/metabolism; Tumor Cells, Cultured; Tumor Suppressor Protein p53/physiology; p38 Mitogen-Activated Protein Kinases; GADD45 Proteins

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Collection: 01-internacional Database: MEDLINE Main subject: Subcellular Fractions / Proteins / G2 Phase / Cyclin B / Mitogen-Activated Protein Kinases / Mitosis Type of study: Risk_factors_studies Limits: Humans Language: En Journal: Oncogene Journal subject: BIOLOGIA MOLECULAR / NEOPLASIAS Year: 2002 Type: Article Affiliation country: United States

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