Transgenic mice producing major histocompatibility complex class II molecules on thyroid cells do not develop apparent autoimmune thyroid diseases.

Li, Yu-Shu; Kanamoto, Naotetsu; Hataya, Yuji; Moriyama, Kenji; Hiratani, Hitomi; Nakao, Kazuwa; Akamizu, Takashi

Li, Yu-Shu; Kanamoto, Naotetsu; Hataya, Yuji; Moriyama, Kenji; Hiratani, Hitomi; Nakao, Kazuwa; Akamizu, Takashi.

Affiliation

Li YS; Translational Research Center, Kyoto University School of Medicine, 54 Shogoin Kawaharacho Sakyo-ku, Kyoto 606-8507, Japan.

Endocrinology ; 145(5): 2524-30, 2004 May.

Article in En | MEDLINE | ID: mdl-14764636

Subject(s)

Autoimmune Diseases/immunology; Histocompatibility Antigens Class II/biosynthesis; Thyroid Diseases/immunology; Thyroid Gland/immunology; Animals; Autoantibodies/biosynthesis; Cell Division; Flow Cytometry; Fluorescent Antibody Technique; Histocompatibility Antigens Class II/genetics; Lymphocyte Activation; Lymphocytes/pathology; Mice; Mice, Inbred C3H; Mice, Transgenic; Promoter Regions, Genetic/genetics; Receptors, Thyrotropin/genetics; T-Lymphocytes/immunology; Thyroid Gland/pathology; Thyroxine/blood

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Collection: 01-internacional Database: MEDLINE Main subject: Autoimmune Diseases / Thyroid Diseases / Thyroid Gland / Histocompatibility Antigens Class II Limits: Animals Language: En Journal: Endocrinology Year: 2004 Type: Article Affiliation country: Japan

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