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The stress response neuropeptide CRF increases amyloid-ß production by regulating γ-secretase activity.
Park, Hyo-Jin; Ran, Yong; Jung, Joo In; Holmes, Oliver; Price, Ashleigh R; Smithson, Lisa; Ceballos-Diaz, Carolina; Han, Chul; Wolfe, Michael S; Daaka, Yehia; Ryabinin, Andrey E; Kim, Seong-Hun; Hauger, Richard L; Golde, Todd E; Felsenstein, Kevin M.
Affiliation
  • Park HJ; Center for Translational Research in Neurodegenerative Disease, Department of Neuroscience, McKnight Brain Institute, College of Medicine, University of Florida, Gainesville, FL, USA Department of Pharmacology and Therapeutics, College of Medicine, McKnight Brain Institute, University of Florida, Ga
  • Ran Y; Center for Translational Research in Neurodegenerative Disease, Department of Neuroscience, McKnight Brain Institute, College of Medicine, University of Florida, Gainesville, FL, USA.
  • Jung JI; Center for Translational Research in Neurodegenerative Disease, Department of Neuroscience, McKnight Brain Institute, College of Medicine, University of Florida, Gainesville, FL, USA.
  • Holmes O; Center for Neurologic Diseases, Brigham and Women's Hospital and Harvard Medical School, Boston, MA, USA.
  • Price AR; Center for Translational Research in Neurodegenerative Disease, Department of Neuroscience, McKnight Brain Institute, College of Medicine, University of Florida, Gainesville, FL, USA.
  • Smithson L; Center for Translational Research in Neurodegenerative Disease, Department of Neuroscience, McKnight Brain Institute, College of Medicine, University of Florida, Gainesville, FL, USA.
  • Ceballos-Diaz C; Center for Translational Research in Neurodegenerative Disease, Department of Neuroscience, McKnight Brain Institute, College of Medicine, University of Florida, Gainesville, FL, USA.
  • Han C; Department of Aging and Geriatric Research, College of Medicine, University of Florida, Gainesville, FL, USA.
  • Wolfe MS; Center for Neurologic Diseases, Brigham and Women's Hospital and Harvard Medical School, Boston, MA, USA.
  • Daaka Y; Department of Anatomy and Cell Biology, College of Medicine, University of Florida, Gainesville, FL, USA.
  • Ryabinin AE; Department of Behavioral Neuroscience, Oregon Health & Science University, Portland, OR, USA.
  • Kim SH; Department of Pharmacology and Therapeutics, College of Medicine, McKnight Brain Institute, University of Florida, Gainesville, FL, USA.
  • Hauger RL; Center of Excellence for Stress and Mental Health, Department of Psychiatry, VA Healthcare System, University of California, San Diego, CA, USA.
  • Golde TE; Center for Translational Research in Neurodegenerative Disease, Department of Neuroscience, McKnight Brain Institute, College of Medicine, University of Florida, Gainesville, FL, USA tgolde@ufl.edu kfelsenstein0@ufl.edu.
  • Felsenstein KM; Center for Translational Research in Neurodegenerative Disease, Department of Neuroscience, McKnight Brain Institute, College of Medicine, University of Florida, Gainesville, FL, USA tgolde@ufl.edu kfelsenstein0@ufl.edu.
EMBO J ; 34(12): 1674-86, 2015 Jun 12.
Article in En | MEDLINE | ID: mdl-25964433
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Stress, Physiological / Corticotropin-Releasing Hormone / Amyloid beta-Peptides / Amyloid Precursor Protein Secretases / Alzheimer Disease / Models, Biological Type of study: Etiology_studies / Prognostic_studies Limits: Animals / Humans Language: En Journal: EMBO J Year: 2015 Type: Article Affiliation country: Gabon
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Stress, Physiological / Corticotropin-Releasing Hormone / Amyloid beta-Peptides / Amyloid Precursor Protein Secretases / Alzheimer Disease / Models, Biological Type of study: Etiology_studies / Prognostic_studies Limits: Animals / Humans Language: En Journal: EMBO J Year: 2015 Type: Article Affiliation country: Gabon