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Tau accumulation induces synaptic impairment and memory deficit by calcineurin-mediated inactivation of nuclear CaMKIV/CREB signaling.
Yin, Yaling; Gao, Di; Wang, Yali; Wang, Zhi-Hao; Wang, Xin; Ye, Jinwang; Wu, Dongqin; Fang, Lin; Pi, Guilin; Yang, Ying; Wang, Xiao-Chuan; Lu, Chengbiao; Ye, Keqiang; Wang, Jian-Zhi.
Affiliation
  • Yin Y; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China;
  • Gao D; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China;
  • Wang Y; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China;
  • Wang ZH; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China;
  • Wang X; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China;
  • Ye J; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China;
  • Wu D; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China;
  • Fang L; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China;
  • Pi G; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China;
  • Yang Y; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China;
  • Wang XC; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China;
  • Lu C; Department of Physiology and Neurobiology, Henan Province Key Laboratory of Brain Research, Xinxiang Medical University, Xinxiang 453003, China;
  • Ye K; Department of Pathology and Laboratory Medicine, Emory University School of Medicine, Atlanta, GA 30322;
  • Wang JZ; Department of Pathophysiology, School of Basic Medicine and the Collaborative Innovation Center for Brain Science, Key Laboratory of Ministry of Education of China for Neurological Disorders, Tongji Medical College, Huazhong University of Science and Technology, Wuhan 430030, China; Co-Innovation Ce
Proc Natl Acad Sci U S A ; 113(26): E3773-81, 2016 06 28.
Article in En | MEDLINE | ID: mdl-27298345
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Synapses / Tau Proteins / Cyclic AMP Response Element-Binding Protein / Calcineurin / Calcium-Calmodulin-Dependent Protein Kinase Type 4 / Alzheimer Disease Type of study: Prognostic_studies Limits: Animals / Humans / Male Language: En Journal: Proc Natl Acad Sci U S A Year: 2016 Type: Article
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Synapses / Tau Proteins / Cyclic AMP Response Element-Binding Protein / Calcineurin / Calcium-Calmodulin-Dependent Protein Kinase Type 4 / Alzheimer Disease Type of study: Prognostic_studies Limits: Animals / Humans / Male Language: En Journal: Proc Natl Acad Sci U S A Year: 2016 Type: Article