Contribution of enhanced transcriptional activation by ER to [12Val] K-Ras mediated NIH3T3 cell transformation.

Kato, K; Ueoka, Y; Kato, K; Hachiya, T; Nishida, J; Wake, N

Kato, K; Ueoka, Y; Kato, K; Hachiya, T; Nishida, J; Wake, N.

Affiliation

Kato K; Department of Reproductive Physiology and Endocrinology, Medical Institute of Bioregulation, Kyushu University, Beppu, Oita, Japan.

Oncogene ; 15(25): 3037-46, 1997 Dec 18.

Article in En | MEDLINE | ID: mdl-9444952

Subject(s)

Cell Transformation, Neoplastic/genetics; Genes, ras/genetics; Receptors, Estrogen/metabolism; Receptors, Progesterone/metabolism; Transcriptional Activation; 3T3 Cells; Animals; Cell Division/drug effects; Gene Expression Regulation, Neoplastic; Mice; Mutation; Oligonucleotides, Antisense/pharmacology; Receptors, Estrogen/drug effects; Receptors, Progesterone/drug effects; Transcription Factors/metabolism; Up-Regulation; ras Proteins/metabolism

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Collection: 01-internacional Database: MEDLINE Main subject: Receptors, Progesterone / Receptors, Estrogen / Transcriptional Activation / Cell Transformation, Neoplastic / Genes, ras Limits: Animals Language: En Journal: Oncogene Journal subject: BIOLOGIA MOLECULAR / NEOPLASIAS Year: 1997 Type: Article Affiliation country: Japan

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