Hypertonicity regulates the function of human neutrophils by modulating chemoattractant receptor signaling and activating mitogen-activated protein kinase p38.

Junger, W G; Hoyt, D B; Davis, R E; Herdon-Remelius, C; Namiki, S; Junger, H; Loomis, W; Altman, A

Junger, W G; Hoyt, D B; Davis, R E; Herdon-Remelius, C; Namiki, S; Junger, H; Loomis, W; Altman, A.

Affiliation

Junger WG; University of California San Diego, Department of Surgery, Division of Trauma, San Diego, California 92103, USA. wjunger@ucsd.edu

J Clin Invest ; 101(12): 2768-79, 1998 Jun 15.

Article in En | MEDLINE | ID: mdl-9637711

Subject(s)

Calcium-Calmodulin-Dependent Protein Kinases/physiology; Mitogen-Activated Protein Kinases; Neutrophil Activation/drug effects; Neutrophil Activation/physiology; Neutrophils/physiology; Receptors, Immunologic/physiology; Receptors, Peptide/physiology; Saline Solution, Hypertonic/pharmacology; Signal Transduction/physiology; Enzyme Activation; Humans; Neutrophils/drug effects; Receptors, Formyl Peptide; Signal Transduction/drug effects; p38 Mitogen-Activated Protein Kinases

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Saline Solution, Hypertonic / Receptors, Immunologic / Signal Transduction / Receptors, Peptide / Calcium-Calmodulin-Dependent Protein Kinases / Neutrophil Activation / Mitogen-Activated Protein Kinases / Neutrophils Limits: Humans Language: En Journal: J Clin Invest Year: 1998 Type: Article Affiliation country: United States

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