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Degradation of Stop Codon Read-through Mutant Proteins via the Ubiquitin-Proteasome System Causes Hereditary Disorders.
Shibata, Norihito; Ohoka, Nobumichi; Sugaki, Yusuke; Onodera, Chiaki; Inoue, Mizuho; Sakuraba, Yoshiyuki; Takakura, Daisuke; Hashii, Noritaka; Kawasaki, Nana; Gondo, Yoichi; Naito, Mikihiko.
Afiliação
  • Shibata N; Division of Molecular Target and Gene Therapy Products, National Institute of Health Sciences, Setagaya-ku, Tokyo 158-8501, Japan.
  • Ohoka N; Division of Molecular Target and Gene Therapy Products, National Institute of Health Sciences, Setagaya-ku, Tokyo 158-8501, Japan.
  • Sugaki Y; Graduate School of Frontier Sciences, University of Tokyo, Kashiwa, Chiba 277-8561, Japan.
  • Onodera C; Graduate School of Frontier Sciences, University of Tokyo, Kashiwa, Chiba 277-8561, Japan.
  • Inoue M; Faculty of Pharmaceutical Sciences, University of Tokyo, Bunkyo-ku, Tokyo 113-0033, Japan.
  • Sakuraba Y; Mutagenesis and Genomics Team, RIKEN BioResource Center, Tsukuba, Ibaraki 305-0074, Japan.
  • Takakura D; Division of Biological Chemistry and Biologicals, National Institute of Health Sciences, Setagaya-ku, Tokyo 158-8501, Japan.
  • Hashii N; Division of Biological Chemistry and Biologicals, National Institute of Health Sciences, Setagaya-ku, Tokyo 158-8501, Japan.
  • Kawasaki N; Division of Biological Chemistry and Biologicals, National Institute of Health Sciences, Setagaya-ku, Tokyo 158-8501, Japan.
  • Gondo Y; Mutagenesis and Genomics Team, RIKEN BioResource Center, Tsukuba, Ibaraki 305-0074, Japan.
  • Naito M; Division of Molecular Target and Gene Therapy Products, National Institute of Health Sciences, Setagaya-ku, Tokyo 158-8501, Japan. Electronic address: miki-naito@nihs.go.jp.
J Biol Chem ; 290(47): 28428-28437, 2015 Nov 20.
Article em En | MEDLINE | ID: mdl-26442586
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Códon de Terminação / Ubiquitina / Complexo de Endopeptidases do Proteassoma / Proteína Reguladora de Apoptosis Semelhante a CASP8 e FADD / Doenças Genéticas Inatas / Mutação Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Revista: J Biol Chem Ano de publicação: 2015 Tipo de documento: Article País de afiliação: Japão
Texto completo
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Códon de Terminação / Ubiquitina / Complexo de Endopeptidases do Proteassoma / Proteína Reguladora de Apoptosis Semelhante a CASP8 e FADD / Doenças Genéticas Inatas / Mutação Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Revista: J Biol Chem Ano de publicação: 2015 Tipo de documento: Article País de afiliação: Japão