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Excessive endosomal TLR signaling causes inflammatory disease in mice with defective SMCR8-WDR41-C9ORF72 complex function.
McAlpine, William; Sun, Lei; Wang, Kuan-Wen; Liu, Aijie; Jain, Ruchi; San Miguel, Miguel; Wang, Jianhui; Zhang, Zhao; Hayse, Braden; McAlpine, Sarah Grace; Choi, Jin Huk; Zhong, Xue; Ludwig, Sara; Russell, Jamie; Zhan, Xiaoming; Choi, Mihwa; Li, Xiaohong; Tang, Miao; Moresco, Eva Marie Y; Beutler, Bruce; Turer, Emre.
Afiliação
  • McAlpine W; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Sun L; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Wang KW; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Liu A; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Jain R; Division of Gastroenterology, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • San Miguel M; Division of Gastroenterology, Department of Internal Medicine, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Wang J; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Zhang Z; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Hayse B; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • McAlpine SG; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Choi JH; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Zhong X; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Ludwig S; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Russell J; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Zhan X; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Choi M; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Li X; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Tang M; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Moresco EMY; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505.
  • Beutler B; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505; bruce.beutler@utsouthwestern.edu emre.turer@utsouthwestern.edu.
  • Turer E; Center for the Genetics of Host Defense, University of Texas Southwestern Medical Center, Dallas, TX 75390-8505; bruce.beutler@utsouthwestern.edu emre.turer@utsouthwestern.edu.
Proc Natl Acad Sci U S A ; 115(49): E11523-E11531, 2018 12 04.
Article em En | MEDLINE | ID: mdl-30442666
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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Proteínas de Transporte / Peptídeos e Proteínas de Sinalização Intracelular / Receptores Toll-Like / Proteína C9orf72 / Inflamação Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Revista: Proc Natl Acad Sci U S A Ano de publicação: 2018 Tipo de documento: Article
Texto completo
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Buscar no Google

Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Proteínas de Transporte / Peptídeos e Proteínas de Sinalização Intracelular / Receptores Toll-Like / Proteína C9orf72 / Inflamação Tipo de estudo: Etiology_studies Limite: Animals Idioma: En Revista: Proc Natl Acad Sci U S A Ano de publicação: 2018 Tipo de documento: Article