UBA52 Attunes VDAC1-Mediated Mitochondrial Dysfunction and Dopaminergic Neuronal Death.

Tiwari, Shubhangini; Singh, Abhishek; Gupta, Parul; K, Amrutha; Singh, Sarika

Tiwari, Shubhangini; Singh, Abhishek; Gupta, Parul; K, Amrutha; Singh, Sarika.

Afiliação

Tiwari S; Division of Toxicology and Experimental Medicine, CSIR-Central Drug Research Institute, Lucknow 226031, India.
Singh A; Division of Toxicology and Experimental Medicine, CSIR-Central Drug Research Institute, Lucknow 226031, India.
Gupta P; Academy of Scientific & Innovative Research (AcSIR), Ghaziabad 201002, India.
K A; Division of Toxicology and Experimental Medicine, CSIR-Central Drug Research Institute, Lucknow 226031, India.
Singh S; Academy of Scientific & Innovative Research (AcSIR), Ghaziabad 201002, India.

ACS Chem Neurosci ; 14(5): 839-850, 2023 03 01.

Article em En | MEDLINE | ID: mdl-36755387

Assuntos

Cálcio; Doença de Parkinson; Animais; Ratos; Apoptose; Cálcio/metabolismo; Neurônios Dopaminérgicos/metabolismo; Potencial da Membrana Mitocondrial; Mitocôndrias/metabolismo; Membranas Mitocondriais/metabolismo; Doença de Parkinson/metabolismo; Canal de Ânion 1 Dependente de Voltagem/metabolismo

Palavras-chave

Parkinson's disease; autophagic cell death; mitochondrial homeostasis; ubiquitin-60S ribosomal protein L40 (UBA52); ubiquitylation; voltage-dependent anion channel 1 (VDAC1)

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Texto completo: 1 Coleções: 01-internacional Base de dados: MEDLINE Assunto principal: Doença de Parkinson / Cálcio Limite: Animals Idioma: En Revista: ACS Chem Neurosci Ano de publicação: 2023 Tipo de documento: Article País de afiliação: Índia

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