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2.
Arkh Anat Gistol Embriol ; 73(11): 101-7, 1977 Nov.
Article Ru | MEDLINE | ID: mdl-597045

Morphological studies in 40 rabbits demonstrated that chronic irritation of the lumbar sympathetic trunks resulted in more pronounced architectonic changes, in vessel walls and in neurons of the lumbar sympathetic ganglia in 15 days than did the bilateral lumbar gangliosympathectomy at the same time. In both series of experiments more prominent changes were noticed in the spinal ganglia than in the spinal cord, that could be explained by different degree of lesions in the nerves supplying them and by organic specification of their angioarchitectonics. The data obtained made it possible to include novocain block (anesthesia) into complex therapy to manage spinal apoplexy in clinic.


Ganglia, Spinal/blood supply , Spinal Cord/blood supply , Sympathetic Nervous System/physiology , Animals , Arteries/pathology , Capillaries/pathology , Rabbits , Sympathectomy , Veins/pathology
3.
Arkh Anat Gistol Embriol ; 73(7): 21-6, 1977 Jul.
Article Ru | MEDLINE | ID: mdl-901227

Modelling of the brain ischemic hypoxia, the hypophysis including, by ligation of the left internal carotid artery in dogs results in weight enlargement of the hypophysis, marked edema of the parenchyma, discomplexity of the cellular elements of the anterior pituitary lobe with morphological signs of hypofunctional condition of the increting cells in the organ studied. Ischemic hypoxia produces lumen augmentation of blood vessels in some areas of the anterior pituitary lobe with resulting increased circulatory capacity in the anterior pituitary lobe. Comparing with the data available in the literature, we conclude that blood circulation manifests a nonspecific reaction to different hypoxic models, while cellular reaction is more specific.


Ischemia/pathology , Pituitary Gland/blood supply , Animals , Blood Vessels/pathology , Capillaries/pathology , Disease Models, Animal , Dogs , Male , Organ Size , Pituitary Gland/pathology , Time Factors
4.
Arkh Anat Gistol Embriol ; 71(12): 22-9, 1976 Dec.
Article Ru | MEDLINE | ID: mdl-141248

The combination of the collateral blood flow in the heart and lungs with effects of Alpine hypoxia and pronounced additional loads was found to allow the detecting of plastical capacities of these organs in a sufficiently full volume. The experiments were performed in 273 dogs by microscopic, macro-microscopic, macroscopic and partly functional methods. The collateral coronary blood flow (after ligation of the anterior interventricular artery) under Alpine conditions (3200 m over the sea level) combined with compensatory hyperfunction of the heart (due to stenosing of the aorta arc), gets worse as compared with the conditions of the valley. In these experiments in mountains the extra- and intraorganic anastomoses are more pronounced, the capacity of the coronary artery branches being less pronounced than in the valley. The muscle fibres grow thicker, the heart weight enlarges, the diffusion distances of capillaries increase and the ratio of the arterial bed capacity and the heart weight decreases. Under Alphine conditions (as compared with the valley) the collateral blood flow of lungs deteriorates (after ligation of two lobar branches of the pulmonary artery or of the lobar vein) against the background of additional loads (stenosing of the aorta arc or pulmonectomy). Deterioration of the collateral bloodflow is related with the combination of conditions of the alphine hypoxia with additional loads resulting in a weakening or even block of compensatory reactions of pulmonary or bronchial arteries and veins.


Altitude Sickness/physiopathology , Capillaries/physiopathology , Collateral Circulation , Coronary Circulation , Coronary Vessels/physiopathology , Hypoxia/physiopathology , Lung/blood supply , Pulmonary Circulation , Altitude Sickness/pathology , Animals , Aortic Valve Stenosis/etiology , Aortic Valve Stenosis/physiopathology , Capillaries/pathology , Cardiomegaly/etiology , Cardiomegaly/physiopathology , Dogs , Elasticity , Hypoxia/etiology
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