La práctica enfermera frente a las agresiones sexuales / Nurse practice against sexual assault

La violencia de género es un problema de gran magnitud, tanto por su frecuencia como por sus graves consecuencias para la salud de las mujeres, y constituye un importante problema de salud pública. Las agresiones sexuales son una de las múltiples formas que adopta la violencia de género. El personal de enfermería tiene un papel central en la atención de los casos de agresiones sexuales agudas. El objetivo del presente artículo es presentar el papel del equipo de enfermería en las agresiones sexuales agudas. Para ello, se realiza toda una serie de recomendaciones tanto desde el rol autónomo como desde el rol de colaboración de enfermería. El gran interés de este artículo radica en que, una vez leído, enfermería, sea del ámbito asistencial que sea, sepa qué hacer y qué no hacer ante un caso de agresión sexual aguda (AU)

Gender violence is a serious problem as well as its frequency and its serious consequences for women health. This fact contributes to a huge public health problem. Gender violence takes on different ways; being sexual assaults is one of these. Nursing staff plays a central role in addressing cases of acute sexual assault. The aim of this article is to present the role of nursing staff in acute sexual assault. Thus, a number of recommendations are made both from nursing’s autonomous role as nursing’s collaboration role. The most interesing issue of this article is the can be used as a guide for nursing staff about what to do in sexual assault cases (AU)

[Nurse Practice Sexual Assault]. / La práctica enfermera frente las agresiones sexuales.

Gender violence is a serious problem as well as its serious consequences for women health. This fact contributes to a huge public health problem. Gender violence takes on different ways; being sexual assaults is one of these. Nursing staff plays a central role in addressing cases of acute sexual assault. The aim of this article is to present the role of nursing staff in acute sexual assault. Thus, a number of recommendations are made both from nursing's autonomous role as nursing's collaboration role. The most interesting issue of this article is that it can be used as a guide for nursing staff about what to do in sexual assault cases.

Gender-specific effects of genetic variants within Th1 and Th17 cell-mediated immune response genes on the risk of developing rheumatoid arthritis.

The present study was conducted to explore whether single nucleotide polymorphisms (SNPs) in Th1 and Th17 cell-mediated immune response genes differentially influence the risk of rheumatoid arthritis (RA) in women and men. In phase one, 27 functional/tagging polymorphisms in C-type lectins and MCP-1/CCR2 axis were genotyped in 458 RA patients and 512 controls. Carriers of Dectin-2 rs4264222T allele had an increased risk of RA (OR = 1.47, 95%CI 1.10-1.96) whereas patients harboring the DC-SIGN rs4804803G, MCP-1 rs1024611G, MCP-1 rs13900T and MCP-1 rs4586C alleles had a decreased risk of developing the disease (OR = 0.66, 95%CI 0.49-0.88; OR = 0.66, 95%CI 0.50-0.89; OR = 0.73, 95%CI 0.55-0.97 and OR = 0.68, 95%CI 0.51-0.91). Interestingly, significant gender-specific differences were observed for Dectin-2 rs4264222 and Dectin-2 rs7134303: women carrying the Dectin-2 rs4264222T and Dectin-2 rs7134303G alleles had an increased risk of RA (OR = 1.93, 95%CI 1.34-2.79 and OR = 1.90, 95%CI 1.29-2.80). Also five other SNPs showed significant associations only with one gender: women carrying the MCP-1 rs1024611G, MCP-1 rs13900T and MCP-1 rs4586C alleles had a decreased risk of RA (OR = 0.61, 95%CI 0.43-0.87; OR = 0.67, 95%CI 0.47-0.95 and OR = 0.60, 95%CI 0.42-0.86). In men, carriers of the DC-SIGN rs2287886A allele had an increased risk of RA (OR = 1.70, 95%CI 1.03-2.78), whereas carriers of the DC-SIGN rs4804803G had a decreased risk of developing the disease (OR = 0.53, 95%CI 0.32-0.89). In phase 2, we genotyped these SNPs in 754 RA patients and 519 controls, leading to consistent gender-specific associations for Dectin-2 rs4264222, MCP-1 rs1024611, MCP-1 rs13900 and DC-SIGN rs4804803 polymorphisms in the pooled sample (OR = 1.38, 95%CI 1.08-1.77; OR = 0.74, 95%CI 0.58-0.94; OR = 0.76, 95%CI 0.59-0.97 and OR = 0.56, 95%CI 0.34-0.93). SNP-SNP interaction analysis of significant SNPs also showed a significant two-locus interaction model in women that was not seen in men. This model consisted of Dectin-2 rs4264222 and Dectin-2 rs7134303 SNPs and suggested a synergistic effect between the variants. These findings suggest that Dectin-2, MCP-1 and DC-SIGN polymorphisms may, at least in part, account for gender-associated differences in susceptibility to RA.