Efectos de la ivabradina en la frecuencia cardiaca y los parámetros hemodinámicos en un modelo porcino de shock cardiogénico / Effects of ivabradine on heart rate and hemodynamic parameters in a swine model of cardiogenic shock

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Cardiogenic shock and cardiac arrest.

Severe heart disease may cause hypotension and hypoperfusion, and ultimately circulation may cease altogether. These two clinical syndromes are cardiogenic shock and cardiac arrest, respectively. This review summarizes the causes and clinical features of each, and describes the treatment options available to clinicians managing patients in cardiogenic shock.

Pleural Mesocestoides and cardiac shock in an obese vervet monkey (Cercopithecus aethiops).

Only one case of infection by tetrathyridia larvae of the tapeworm genus Mesocestoides was detected in 416 necropsies of captive vervet monkeys (Cercopithecus aethiops). Two hundred nine larvae were distributed between both pleural cavities. Mass and size ranges of larvae were determined. A plasma cell reaction indicated a humoral immune response to parasite antigens, which may have contributed to acute, lethal cardiac shock. Coagulative myocytolysis was confirmed. The history of this case and associated circumstantial evidence and reports in the literature suggest that infection of primates by tetrathyridia probably occurs after capture rather than before.

The unified concept of shock.

In summary, the shock syndrome has a variety of causes although the same net physiologic insult results. The disease is a deficiency in tissue metabolism brought about by decreased capillary blood flow and cellular hypoxia. The therapeutic regimen employed in any given patient is dectated by the initial cause of shock as well as the physiologic changes that have occurred in the patient. Fluid and electrolyte therapy is the primary method of treatment. Modification of solutions and use of additional drugs may be indicated.