ABSTRACT
Background: The trait-impulsivity hypothesis posits impulsivity as the underlying substrate of Attention-deficit/hyperactivity disorder (ADHD) and Oppositional defiant disorder (ODD) symptom expressions. The current study applied network analysis to evaluate the inter-relationships of dimensions within ADHD (inattention and hyperactivity/impulsivity) and ODD (anger/irritable, vindictiveness, and argumentative/defiant behavior) with components of impulsivity as captured by the UPPS-P model (negative urgency, lack of premeditation, lack of perseverance, sensation seeking, and positive urgency). Method: A total of 324 emerging adults (women = 246) from the general community completed questionnaires covering these dimensions. Results: Our findings showed that the ADHD and ODD dimensions were associated differentially with different types of impulsivity, in their unique patterns of network connectivities, a possibility that has had little attention in the trait-impulsivity hypothesis literature. Conclusions: This study is the first to tease out the unique associations of the ADHD and ODD dimensions with different types of impulsivity, and in that way provide new contributions to our understanding of the existing trait impulsivity theory. Our findings would be especially relevant to those interested in understanding how different dimensions of trait impulsivity underly the ADHD and ODD dimensions.
ABSTRACT
Sexual offender risk assessment practice is considered by many to be atheoretical. The identification of the most predictive risk factors and tools has typically overshadowed questions about etiology. To gain insight into the origins of criminal behavior among sexual offenders, we developed and validated an etiological model of risk based on the theoretical framework of Beech and Ward. Our model focused on persistence rather than onset, and encompassed both the sexual and nonsexual criminal activity of these offenders. It comprised two pathways. The first was characterized by sexual victimization, social isolation, and early deviant sexual fantasies. It led to a prolific involvement in sexual criminality (especially toward children) and predicted sexual recidivism. The second pathway was characterized by externalization problems, sexual promiscuity, and physical/psychological victimization, and was associated with nonsexual offending and serious sexual offenses directed (mostly) toward women. It predicted all types of recidivism.
Subject(s)
Criminals/psychology , Models, Psychological , Recidivism , Sex Offenses/psychology , Humans , Risk Assessment , Risk FactorsABSTRACT
The trait-impulsivity etiological model assumes that a general factor (trait-impulsivity) underlies attention-deficit/hyperactivity disorder (ADHD), oppositional defiant disorder (ODD), and other externalizing disorders. We investigated the plausibility of this assumption by testing the factor structure of ADHD and ODD in a bifactor framework for a clinical sample of 1420 children between 6 and 18 years of age (M = 9.99, SD = 3.34; 85% male). Further, the trait-impulsivity etiological model assumes that ODD emerges only if environmental risk factors are present. Our results support the validity of the trait-impulsivity etiological model, as they confirm that ADHD and ODD share a strong general factor of disruptive behavior (DB) in this clinical sample. Furthermore, unlike the subdimensions of ADHD, we found that the specific ODD factor explained as much true score variance as the general DB factor. This suggests that a common scale of ADHD and ODD may prove to be as important as a separate ODD subscale to assess externalizing problems in school-age children. However, all other subscales of ADHD may not explain sufficient true score variance once the impact of the general DB factor has been taken into consideration. In accordance with the trait-impulsivity model, we also showed that all factors, but predominantly the general factor and specific inattention factor, predicted parent-rated impairment, and that predominantly ODD and impulsivity are predicted by environmental risk factors.
Subject(s)
Attention Deficit Disorder with Hyperactivity/etiology , Attention Deficit and Disruptive Behavior Disorders/etiology , Impulsive Behavior/physiology , Personality/physiology , Adolescent , Attention Deficit Disorder with Hyperactivity/psychology , Attention Deficit and Disruptive Behavior Disorders/psychology , Child , Female , Humans , Male , Models, Psychological , Problem Behavior/psychology , Social EnvironmentABSTRACT
BACKGROUND: The aim of this study was to investigate the cortical and subcortical brain structures in Parkinson's disease (PD) with visual hallucination (VH), and to elucidate the association between the proposed hypothesis of VH in PD and regional brain volume changes. METHODS: We used 3T magnetic resonance imaging (MRI) and voxel-based morphometry (VBM) to investigate the brain structures of PD patients with VH (PD-VH; n = 13) and PD patients without VH (PD-C; n = 13). RESULTS: The PD-VH patients showed significant cortical atrophy compared to the PD-C patients in the bilateral dorsolateral prefrontal cortex, left rostral region of the prefrontal cortex, left ventral section of the cingulate cortex, bilateral primary visual cortex, and secondary visual cortex including the left inferior occipital gyrus, right lingual cortex, right supramarginal gyrus, and left fusiform gyrus. Significant subcortical atrophic changes were observed in the white matter of the right parahippocampal gyrus, the bilateral posterior part of the cingulate gyrus, the left lingual gyrus, and the right middle occipital gyrus. CONCLUSIONS: VH in PD can occur due to distinctive neuroanatomical involvement.
Subject(s)
Brain/pathology , Hallucinations/pathology , Parkinson Disease/pathology , Aged , Atrophy/complications , Atrophy/pathology , Female , Hallucinations/complications , Humans , Image Processing, Computer-Assisted , Magnetic Resonance Imaging , Male , Middle Aged , Parkinson Disease/complicationsABSTRACT
En los últimos años, la investigación de los recursos psicológicos que favorecen la salud y el bienestar han experimentado un aumento considerable. Recientes datos muestran a partir de estudios altamente rigurosos y controlados, algunos de los beneficios que producen los estados y afectos positivos, como también del optimismo y el bienestar, entre otros estados positivos en la salud física y psicológica. A pesar de esta evidencia, existe aún poca información de los mecanismos y variables relacionadas en los procesos de bienestar, optimismo y afectos positivos. Así mismo existe escaso consenso acerca de los mecanismos psicológicos y/o neurofisiológicos que expliquen la relación con los estados positivos y su beneficio para la salud y principalmente acerca de las variables causales o etiológicas del bienestar. Se revisa la evidencia científica acerca de los beneficios de los estados positivos como el optimismo, felicidad y principalmente el bienestar. Se propone un modelo causal conceptual que plantea que el nivel de bienestar subjetivo y psicológico, está influenciado previamente por el nivel de optimismo. De esta forma y basada en la literatura y evidencia previa se plantea que el optimismo, al ser una condición aprendida desde la niñez, favorecería la mayor cantidad de experiencias positivas y emociones positivas. Estos estados afectivos positivos mediarían la relación entre el optimismo y el bienestar y sus consecuencias.
Research into the psychological resources that benefit health and well-being have increased dramatically in recent years. The latest data from highly rigorous and controlled studies show some of the benefits that produce positive states and affects, as well as optimism and well-being, among other positive states in physical and psychological health. In spite of this evidence, there is little information about the mechanisms and variables related to the processes of well-being, optimism and positive affects. In addition, there is no real consensus regarding the psychological and/or neurophysiological mechanisms that explain the relation to positive states and their benefit for health and mainly with respect to the causal or etiological variables of well-being. The scientific evidence about the benefits of positive states like optimism, happiness and mainly well-being is reviewed. A conceptual causal model is proposed which suggests that the level of subjective and psychological well-being is previously influenced by the level of optimism. This way, and based on the prior literature and evidence, it is proposed that optimism, being a condition learned from childhood, may stimulate the greatest number of positive experiences and positive emotions. These positive affect states would then mediate the relation between optimism and well-being and their consequences.