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1.
J Lipid Res ; 55(5): 860-9, 2014 May.
Article in English | MEDLINE | ID: mdl-24616481

ABSTRACT

Recently it has been shown that levels of circulating oxidized LDL immune complexes (ox-LDL-ICs) predict the development of diabetic retinopathy (DR). This study aimed to investigate whether ox-LDL-ICs are actually present in the diabetic retina, and to define their effects on human retinal pericytes versus ox-LDL. In retinal sections from people with type 2 diabetes, costaining for ox-LDL and IgG was present, proportionate to DR severity, and detectable even in the absence of clinical DR. In contrast, no such staining was observed in retinas from nondiabetic subjects. In vitro, human retinal pericytes were treated with native LDL, ox-LDL, and ox-LDL-IC (0-200 mg protein/l), and measures of viability, receptor expression, apoptosis, endoplasmic reticulum (ER) and oxidative stresses, and cytokine secretion were evaluated. Ox-LDL-IC exhibited greater cytotoxicity than ox-LDL toward retinal pericytes. Acting through the scavenger (CD36) and IgG (CD64) receptors, low concentrations of ox-LDL-IC triggered apoptosis mediated by oxidative and ER stresses, and enhanced inflammatory cytokine secretion. The data suggest that IC formation in the diabetic retina enhances the injurious effects of ox-LDL. These findings offer new insights into pathogenic mechanisms of DR, and may lead to new preventive measures and treatments.


Subject(s)
Antigen-Antibody Complex , Capillaries/pathology , Diabetes Mellitus, Type 2/immunology , Lipoproteins, LDL/toxicity , Pericytes/drug effects , Retina/physiopathology , Aged , Apoptosis/drug effects , Cell Survival/drug effects , Cytokines/metabolism , Endoplasmic Reticulum Stress/drug effects , Humans , Immunoglobulin G/metabolism , Intracellular Space/drug effects , Intracellular Space/metabolism , Lipoproteins, LDL/metabolism , Pericytes/metabolism , Pericytes/pathology , Protein Transport/drug effects , Reactive Oxygen Species/metabolism , Receptors, IgG/metabolism , Receptors, Scavenger/metabolism
2.
Clin Immunol ; 139(3): 258-66, 2011 Jun.
Article in English | MEDLINE | ID: mdl-21439910

ABSTRACT

Diabetic nephropathy is characterized by progressive mesangial expansion. Although we have reported that circulating oxidized LDL-containing immune complexes (oxLDL-IC) are associated with abnormal levels of albuminuria, the underlying mechanisms have not been investigated. In this study, we have studied the effect of oxLDL-IC on collagen IV expression by mesangial cells. We found that oxLDL-IC markedly stimulated collagen IV expression in a concentration- and time-dependent fashion while oxLDL only had moderate effect. We also found that oxLDL-IC stimulated collagen IV expression by engaging Fc gamma receptor (FcγR) I and III, but not FcγRII, and that p38 MAPK, JNK and PKC pathways were involved in collagen IV expression. Furthermore, we found that oxLDL-IC stimulated FcγRI expression, suggesting a positive feedback mechanism involved in oxLDL-IC-stimulated collagen IV expression. Taken together, this study showed that oxLDL-IC stimulated collagen IV in mesangial cells via FcγRI and FcγRIII, and the expression of FcγRI was increased by oxLDL-IC.


Subject(s)
Antigen-Antibody Complex/immunology , Collagen Type IV/biosynthesis , Glomerular Mesangium/immunology , Lipoproteins, LDL/immunology , Mesangial Cells/immunology , Receptors, IgG/genetics , Receptors, IgG/immunology , Collagen Type IV/immunology , Diabetic Nephropathies/immunology , Flow Cytometry , Glomerular Mesangium/cytology , Glomerular Mesangium/drug effects , Humans , Immunoblotting , Mesangial Cells/drug effects , Mesangial Cells/metabolism , Mitogen-Activated Protein Kinases/antagonists & inhibitors , Mitogen-Activated Protein Kinases/immunology , Protein Kinase Inhibitors/pharmacology , RNA/chemistry , RNA/genetics , Reverse Transcriptase Polymerase Chain Reaction
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