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1.
Zootaxa ; 4895(2): zootaxa.4895.2.1, 2020 Dec 15.
Article in English | MEDLINE | ID: mdl-33756900

ABSTRACT

In this study, we designate lectotypes and fix the identities of two Australian Castniidae species, Synemon sophia (White, 1841), the type species of Synemon Doubleday, 1846, and Synemon parthenoides R. Felder, 1874. Furthermore, we describe seven new species of Castniidae from Western Australia, two new species of the Synemon sophia group, Synemon anthracica Kallies Edwards sp. n. and Synemon crocea Kallies Edwards sp. n., three species in the Synemon magnifica Strand, 1911 group, Synemon semaphora Kallies Edwards sp. n., Synemon angustiptera Kallies Edwards sp. n. and Synemon petrophila Kallies Edwards sp. n., one new species of unclear affinity, Synemon cacumina Kallies Edwards sp. n., and one new species related to Synemon directa Westwood, 1877, Synemon victoriae Kallies Edwards sp. n. We report Restionaceae, a host-plant family not previously recorded for Castniidae.


Subject(s)
Lepidoptera , Moths , Animals , Australia , Plants
2.
Congenit Anom (Kyoto) ; 51(2): 80-6, 2011 Jun.
Article in English | MEDLINE | ID: mdl-21039913

ABSTRACT

The objective of the present study was to estimate the preventive effect of folic acid for structural birth defects (i.e. congenital abnormalities [CAs]) in the offspring of pregnant women with diabetes mellitus type 1 (DM-1). The occurrence of medically recorded DM-1 in pregnant women who had malformed fetuses/newborns (cases) and delivered healthy babies (controls) with or without folic acid supplementation was compared in the population-based Hungarian Case-Control Surveillance System of Congenital Abnormalities. The case group included 22,843 offspring, and there were 79 (0.35%) pregnant women with DM-1, while the control group comprised of 38,151 newborns, and 88 (0.23%) had mothers with DM-1. Case mothers with DM-1 associated with a higher risk of total rate of CAs in their offspring (OR with 95% CI: 1.5, 1.1-2.0) compared to the total rate of CAs in the offspring of non-diabetic case mothers. This higher risk can be explained by four specific types/groups of CAs: isolated renal a/dysgenesis; obstructive CA of the urinary tract; cardiovascular CAs; and multiple CAs, namely caudal dysplasia sequence. However, there was no higher rate of total CAs in the children of pregnant women with DM-1 after folic acid supplementation; in addition, neural-tube defect and renal a/dysgenesis did not occur. However, this benefit cannot be explained by the CA reduction effect of folic acid during the critical period of major CAs. In conclusion, there was a certain reduction in maternal teratogenic effect of DM-1 after folic acid supplementation during pregnancy, but the explanation of this effect requires further study.


Subject(s)
Congenital Abnormalities/prevention & control , Diabetes Mellitus, Type 1/complications , Dietary Supplements , Folic Acid/administration & dosage , Pregnancy Complications/prevention & control , Adult , Case-Control Studies , Congenital Abnormalities/epidemiology , Congenital Abnormalities/etiology , Diabetes Mellitus, Type 1/epidemiology , Female , Humans , Hungary/epidemiology , Infant, Newborn , Population Surveillance , Pregnancy , Pregnancy Complications/epidemiology , Pregnancy Complications/etiology
4.
Vet Rec ; 100(2): 36, 1977 Jan 08.
Article in English | MEDLINE | ID: mdl-836607
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