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1.
Affect Sci ; 3(2): 400-405, 2022 Jun.
Article in English | MEDLINE | ID: mdl-36046002

ABSTRACT

While material from waking life is often represented in dreams, it is less clear whether and how dreams impact waking life. Here, we assessed whether dream mood and content from home diaries predict subsequent waking mood using both subjective self-reports and an objective automated word detection approach. Subjective ratings of dream and morning mood were highly correlated within participants for both negative and positive valence, suggesting that dream mood persists into waking. Text analyses revealed similar relationships between affect words in dreams and morning mood. Moreover, dreams referencing death or the body were related to worse morning mood, as was first-person singular pronoun usage (e.g., "I"). Dreams referencing leisure or ingestion, or including first-person plural pronouns (e.g., "we"), were related to better morning mood. Together, these results suggest that subjective experiences during sleep, while often overlooked, may be an important contributor to waking mood. Supplementary Information: The online version contains supplementary material available at 10.1007/s42761-021-00080-8.

2.
Case Rep Neurol ; 12(2): 255-259, 2020.
Article in English | MEDLINE | ID: mdl-32884544

ABSTRACT

Mutations in the myelin protein zero (MPZ) gene can cause a variety of clinical and electrophysiological forms of genetic neuropathies including Charcot-Marie-Tooth (CMT) type 1B disease which is characterized by demyelinating features. We present a father and daughter with neuropathy carrying a novel 31 base pair duplication mutation in the 5' untranslated region of the MPZ gene, c.-23_8dup31. Genetic analysis and protein modeling indicated that this is a frameshift mutation resulting in premature truncation of the encoded protein. The daughter underwent repeat neurological examination and electromyography testing over an 11-year time span demonstrating no clinical or electrophysiological change. Our study expands the clinical and genetic spectrum of mutations that can cause CMT type 1B disease and demonstrates the value of sequence analysis of noncoding portions of a gene that are not intronic.

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