ABSTRACT
We describe a 33-year-old male with angina pectoris who was found to have abnormal origin and course of right coronary artery from left aortic cusp. The abnormal origin was identified by conventional coronary angiography but the abnormal course of the vessel between ascending aorta and the main pulmonary artery was precisely delineated by high resolution CT angiography. Patient underwent successful surgical transposition of the anomalous vessel with complete resolution of symptoms.
Subject(s)
Coronary Angiography/methods , Coronary Vessel Anomalies/diagnostic imaging , Tomography, X-Ray Computed , Adult , Humans , MaleABSTRACT
Anomalous coronary arteries are a rare but recognized cause of myocardial ischemia and sudden death. Most patients with coronary anomalies remain asymptomatic because the anomaly either does not produce any symptoms during life or the first manifestation is sudden death. Noninvasive imaging plays crucial role in diagnosis of the potentially life threatening coronary anomalies.
Subject(s)
Coronary Vessel Anomalies/diagnosis , Magnetic Resonance Angiography/methods , Tomography, X-Ray Computed/methods , Coronary Vessel Anomalies/diagnostic imaging , HumansABSTRACT
Angiotensin converting enzyme inhibitors are one of the most commonly prescribed medications and angioedema of upper aerogastric tracts is a well recognized complication. Isolated visceral angioedema with the use of angiotensin converting enzyme inhibitors is rare and is relatively under diagnosed. The visceral angioedema should be considered in patients taking angiotensin converting enzyme inhibitors who develop gastrointestinal complaints. We report a case of subacute intestinal obstruction from the use of benazepril, which was promptly resolved after withdrawing benazepril.
Subject(s)
Angioedema/chemically induced , Angioedema/diagnosis , Angiotensin-Converting Enzyme Inhibitors/adverse effects , Benzazepines/adverse effects , Adult , Diagnosis, Differential , Female , Humans , Hypertension/complications , Hypertension/drug therapy , Intestinal Diseases/diagnosis , VisceraABSTRACT
We report a case of aortic valve endocarditis with aortic root abscess from Candida parapsilosis occurring 8 months after transient candidemia. Despite the fact that the patient was treated appropriately, candidemia persisted and later on presented with an embolic stroke as a complication of fungal endocarditis.
Subject(s)
Blood/microbiology , Candida/isolation & purification , Candidiasis/diagnosis , Candidiasis/microbiology , Endocarditis/diagnosis , Endocarditis/microbiology , Aortic Valve/microbiology , Candidiasis/complications , Candidiasis/therapy , Embolism/diagnosis , Embolism/etiology , Embolism/therapy , Endocarditis/complications , Endocarditis/therapy , Humans , Male , Middle Aged , Stroke/diagnosis , Stroke/etiology , Stroke/therapy , Time Factors , Treatment OutcomeABSTRACT
Cardiac action potential is affected by extracellular gradients in potassium, calcium, and magnesium with resultant electrocardiographic changes varying from subtle findings to marked repolarization abnormalities. This report presents marked electrocardiographic changes resulting from a combination of electrolytes depletion in a patient with bulimia and anorexia nervosa.
Subject(s)
Anorexia Nervosa/complications , Anorexia Nervosa/physiopathology , Bulimia/complications , Bulimia/physiopathology , Electrocardiography , Electrolytes/metabolism , Heart/physiopathology , Adult , Anorexia Nervosa/metabolism , Bulimia/metabolism , Female , HumansABSTRACT
Cardiac troponin I is released from myocytes in both reversible and irreversible myocardial injury. The changes in myocyte membrane permeability resulting from the injury could be enough for the release of cardiac troponins from the free cytosolic pool of myocytes without structural damage. We report a case of parvovirus B19 myocarditis in a 26-year-old male who developed regional wall motion abnormalities and severe left ventricular systolic dysfunction with elevated serum levels of cardiac troponin I (peak=11.7 ng/ml). Diagnosis of parvovirus myocarditis was confirmed by presence of high titers of parvovirus B19 IgG and identification of parvovirus B19 DNA by polymerase chain reaction. Within a few days of supportive treatment, the regional wall motion abnormalities resolved, the cardiac function recovered, and the elevation in serum cardiac troponin I subsided. This case further denotes the possibility of release of cardiac troponin I in non-ischemic, reversible myocardial injury.