ABSTRACT
BACKGROUND: Data on the effects of weight training on joint morphology are mostly restricted to muscle gain. However, in many circumstances, it is not stated if there are negative consequences for the joints and their surrounding components. This study was conducted to explore whether long-term excessive resistance training (RT) causes hidden pathological alterations in the shoulder. METHODS: A total of eleven asymptomatic sportsmen (22 shoulders) underwent clinical and radiological examination of both shoulder joints. All participants had engaged in bodybuilding for at least four years, at least three times per week, and for at least four hours per week. All participants were examined clinically using the Constant Murley Score (CMS), Simple Shoulder Test (SST), UCLA Activity Test, and a specially designed questionnaire. All participants received a bilateral shoulder MRI. The MRI scans were checked for pathology using a checklist. RESULTS: Maximum scores were observed for the SST and UCLA Activity Test. The CMS was 97.7 points on average (range, 87-100). RT had been conducted for a mean of 10.7 years (range, 4-20), for an average of 8.8 h a week (range, 4-12). MRI examinations revealed two supraspinatus tendinopathies (9.1%), one labral change (4.5%), three humeral tuberosity cysts (13.6%), fourteen acromioclavicular (AC) joint hypertrophies (63.6%), five AC joint osteophytes (22.7%), and ten signs of AC joint inflammation (45.5%). CONCLUSIONS: The research results show that strength is associated with MRI-documented AC joint pathology. However, it appears that RT may not negatively affect other anatomical structures of the shoulder.
ABSTRACT
Background: Glenoid wear is a common complication of anatomical total shoulder arthroplasty (aTSA) with a metal-backed glenoid (MBG), and the clinical and radiological results of historical implants are poor. The aim of this work was to evaluate the clinical and radiological results of 25 participants as well as the longevity after implantation of an anatomic shoulder prosthesis with a recent, modular cementless flat metal-backed glenoid component after a mean follow-up of 5.7 years. Methods: Clinically, the Simple Shoulder Test (SST), UCLA Activity Score (UCLA), and Constant Murley Score (CMS) were evaluated. Radiographically, the radiolucent lines (RLs), humeral head migration (HHM), and lateral glenohumeral offset (LGHO) were assessed. Survival was calculated with Kaplan−Meier curves and life-table analysis. Results: The mean CMS at follow-up was 46.2 points (range: 14−77; SD: 19.5). In terms of the SST score, the average value was 6.5 points (range: 1−10; SD: 3.5). The UCLA activity score showed a mean value of 5.9 points (range: 1−9; SD: 2.1). There were 17 revisions after a mean follow-up of 68.2 months (range: 1.8−119.6; SD: 27.9). HHM occurred in every patient, with a mean measurement of 6.4 mm (range: 0.5−13.4; SD: 3.9; p < 0.0001). The mean LGHO between the initial postoperative and follow-up images was 2.6 mm (range: 0−4.0; SD: 1.5; p < 0.0001). RLs were found in 22 patients (88%) around the glenoid and in 21 patients (84%) around the humeral head prosthesis. Conclusion: The clinical and radiographic outcomes after metal-backed glenoids were poor at 2.2 to 8.4 years of follow-up. We determined devastating survival in the majority of cases (68%), with mostly inlay wear (71%) as the main reason that led to revision surgery. The use of metalback genoids cannot be recommended based on the data of this study.
ABSTRACT
We investigated if a chronic, enhanced immunosuppressed condition, beyond the immunodeficiency related to diabetes, is associated with clinical failures after combined surgical and medical treatment for diabetic foot infection (DFI). This is a case-control cohort study in a tertiary centre for diabetic foot problems, using case-mix adjustments with multivariate Cox regression models. Among 1013 DFI episodes in 586 patients (median age 67 years; 882 with osteomyelitis), we identified a chronic, enhanced immune-suppression condition in 388 (38%) cases: dialysis (85), solid organ transplantation (25), immune-suppressive medication (70), cirrhosis (9), cancer chemotherapy (15) and alcohol abuse (243). Overall, 255 treatment episodes failed (25%). By multivariate analysis, the presence (as compared with absence) of chronic, enhanced immune-suppression was associated with a higher rate of clinical failures in DFI cases (hazard ratio 1.5, 95% confidence interval 1.1-2.0). We conclude that a chronic, enhanced immune-suppressed state might be an independent risk factor for treatment failure in DFI. Validation of this hypothesis could be useful information for both affected patients and their treating clinicians.