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J Pediatr ; 149(1): 89-97, 2006 Jul.
Article in English | MEDLINE | ID: mdl-16860134

ABSTRACT

OBJECTIVE: To conduct an open-label, multinational, multicenter study examining the safety and efficacy of recombinant human acid alpha-glucosidase (rhGAA) in treatment of infantile-onset Pompe disease. STUDY DESIGN: We enrolled 8 infant patients who had Pompe disease with GAA activity <1% of normal, cardiomyopathy, and hypotonia. In the 52-week initial phase, rhGAA was infused intravenously at 10 mg/kg weekly; an extension phase continued survivors' treatment with 10 to 20 mg/kg of rhGAA weekly or 20 mg/kg every 2 weeks for as long as 153 weeks. Safety measurements included adverse events, laboratory tests, and anti-rhGAA antibody titers. Efficacy evaluations included survival, ventilator use, echocardiograms, growth, and motor and cognitive function. RESULT: After 52 weeks of treatment, 6 of 8 patients were alive, and 5 patients were free of invasive ventilator support. Clinical improvements included ameliorated cardiomyopathy and improved growth and cognition. Five patients acquired new motor milestones; 3 patients walked independently. Four patients died after the initial study phase; the median age at death or treatment withdrawal for all patients was 21.7 months, significantly later than expected for patients who were not treated. Treatment was safe and well tolerated; no death was drug-related. CONCLUSION: rhGAA improved ventilator-free survival, cardiomyopathy, growth, and motor function in patients with infantile-onset Pompe disease compared with outcomes expected for patients without treatment.


Subject(s)
Glycogen Storage Disease Type II/drug therapy , Glycogen Storage Disease Type II/mortality , alpha-Glucosidases/therapeutic use , Body Height/drug effects , Body Weight/drug effects , Cardiomyopathy, Hypertrophic/drug therapy , Cardiomyopathy, Hypertrophic/etiology , Child Development , Europe/epidemiology , Female , Glycogen/metabolism , Glycogen Storage Disease Type II/complications , Hearing Disorders/etiology , Humans , Infant , Infant, Newborn , Infusions, Intravenous , Male , Muscle Hypotonia/drug therapy , Muscle Hypotonia/etiology , Muscle, Skeletal/metabolism , Muscle, Skeletal/pathology , Respiration, Artificial , Treatment Outcome , United States/epidemiology , alpha-Glucosidases/metabolism
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