Mechanism of Dyspnea during Exercise in Children with Corrected Congenital Heart Disease.

This study will evaluate cardiorespiratory and peripheral muscle function and their relationship with subjective dyspnea threshold after the surgical correction of congenital heart disease in children. Thirteen children with surgically repaired congenital heart disease were recruited. Each participant performed an incremental exercise test on a cycle ergometer until exhaustion. Gas exchanges were continuously sampled to measure the maximal aerobic parameters and ventilatory thresholds. The functional capacity of the subjects was assessed with a 6 min walk test. At the end of the exercise test, isokinetic Cybex Norm was used to evaluate the strength and endurance of the knee extensor muscle in the leg. Dyspnea was subjectively scored with a visual analog scale during the last 15 s of each exercise step. Oxygen consumption measured at the dyspnea score/VO2 relationship located at the dyspnea threshold, at which dyspnea suddenly increased. Results: The maximal and submaximal values of the parameters describing the exercise and the peripheral muscular performances were: VO2 Peak: 33.8 ± 8.9 mL·min-1·kg-1; HR: 174 ± 9 b·min-1; VEmax: 65.68 ± 15.9 L·min-1; P max: 117 ± 27 W; maximal voluntary isometric force MVIF: 120.8 ± 41.9 N/m; and time to exhaustion Tlim: 53 ± 21 s. Oxygen consumption measured at the dyspnea threshold was related to VO2 Peak (R2 = 0.74; p < 0.01), Tlim (R2 = 0.78; p < 0.01), and the distance achieved during the 6MWT (R2 = 0.57; p < 0.05). Compared to the theoretical maximal values for the power output, VO2, and HR, the surgical correction did not repair the exercise performance. After the surgical correction of congenital heart disease, exercise performance was impeded by alterations of the cardiorespiratory function and peripheral local factors. A subjective evaluation of the dyspnea threshold is a reliable criterion that allows the prediction of exercise capacity in subjects suffering from congenital heart disease.

Warm-up durations in a hot-dry climate affect thermoregulation, mean power-output and fatigue, but not peak power in specific soccer repeated-sprint ability.

BACKGROUND: This study addressed the lack of data on the effect of warm-up (WU) duration in hot-dry climate (~ 30 °C; ~ 18% RH), on thermoregulation, muscular power-output, and fatigue after specific soccer repeated-sprint test (RSA). METHODS: Eleven amateur soccer players participated in a cross-over randomized study and they underwent the Bangsbo repeated-sprint test, after three WU durations (i.e. WU10, WU15 and WU20 min) at 70% of MAV, and on different days. Peak power (PP), mean power (MP) and the fatigue index (FI) were recorded and analyzed. Likewise, heart rate (HR), tympanic temperature (Ttym), mean body temperature (MBT) and rating of perceived exertion (RPE) were recorded during each session. RESULTS: The repeated measure ANOVA showed that MP improved after WU15 in comparison to WU10 and WU20 (p = 0.04 and p = 0.001; respectively). Nonetheless, no significant effect on PP was recorded after all WU durations. FI during RSA increased after WU20 in comparison to WU15 and WU10 (p < 0.001 and p = 0.003; respectively). Higher RPE values (p < 0.001) were recorded after WU15 and WU20 in comparison to WU10 duration. The two-way ANOVA showed higher ΔTtym and ΔMBT values after WU15 and WU20 compared to WU10 (p = 0.039 and p < 0.001for Ttym; p = 0.005 and p < 0.001 for MBT, respectively). CONCLUSIONS: The WU15 at 70% of MAV better assists mean power-output during soccer RSA in hot-dry (~ 30 °C; 18% RH) climate, but not peak power. Reducing WU duration up to 10 min seems to be insufficient to induce beneficial physiological changes necessary for optimizing repeated-sprint performance, while its extension up to 20 min remains detrimental for muscular power and induces higher fatigue.