One-pot synthesis of interfacially bonded Bi4O5Br2/Bi2S3 Z-scheme heterostructures with boosted photocatalysis towards dodecylbenzenesulfonate and real hotel laundry wastewater.

The ubiquitous contamination of surfactants in wastewater has raised global concerns. Photocatalysis is deemed as a promising yet challenging approach for the decomposition of surfactant residues. Herein, a novel Z-scheme heterojunction of Bi4O5Br2/Bi2S3 with covalent S-O bonds was prepared via a facile one-pot hydrothermal and subsequent annealing process. The prepared optimal Bi4O5Br2/Bi2S3 composite exhibited remarkable photo-degradation activity towards the sodium dodecylbenzene sulfonate (SDBS). The Z-scheme reaction mechanism was proposed and validated by meticulous analysis of quenching tests, ESR spectroscopy and DFT calculations. Furthermore, the presence of chemical S-O linkages between Bi4O5Br2 and Bi2S3 was identified via FT-IR and XPS analyses, which served as a distinct bridge to modify the Z-scheme route for carrier transport. The Z-scheme heterostructure, in conjunction with chemical S-O bonds, synergistically enhanced the separation rate of electron-hole pairs and thus greatly boosted the photocatalytic activity. Additionally, the possible degradation pathways of SDBS were proposed by using HR-MS technology. Moreover, real hotel laundry wastewater could be efficiently disposed by the photocatalysis of the Bi4O5Br2/Bi2S3 with a decrease in the COD value from 428 to 74 mg/L, indicating that the fabricated Z-scheme heterojunction hold great promise for effectively removing refractory surfactant contaminants from aquatic environment.

Design and Fabrication of a Novel Corona-Shaped Metamaterial Biosensor for Cancer Cell Detection.

The early detection and diagnosis of cancer presents significant challenges in today's healthcare. So, this research, suggests an original experimental biosensor for cell cancer detection using a corona-shaped metamaterial resonator. This resonator is designed to detect cancer markers with high sensitivity, selectivity, and linearity properties. By exploiting the unique properties of the corona metamaterial structure in the GHz regime, the resonator provides enhanced interaction of electromagnetic waves and improved detection skills. Through careful experimental, simulation, and optimization studies, we accurately demonstrate the resonator's ability to detect cancer. The proposed detection system is capable of real-time non-invasive cancer detection, allowing for rapid intervention and better patient outcomes. The sensitivity value was confirmed through simulation, estimated at 0.1825 GHz/RIU. The results of two different simulation methods are used: the simulation software CST Studio Suite (version 2017) based on the finite element method (FEM), and the simulation software ADS (version 2019) based on the equivalent circuit method, thereby increasing confidence in the convergence of simulation and measurement results. This work opens new avenues for developing advanced detection technologies in the field of oncology, and paves the way for more effective cancer diagnosis. The experimental study verified that this realized sensor has very small frequency shifts, significantly small electrical dimension and miniaturization, high sensitivity, and good linearity. The suggested configurations showed a capacity for sensing cancer cells in the GHz regime.

Metabolic, Apoptotic and Fibro-Inflammatory Profiles of the Heart Exposed to Environmental Electromagnetic Fields.

Environmental stress can disturb the integrative functioning of the cardiovascular system and trigger a number of adaptive and/or maladaptive cell responses. Concomitant with the expanding use of mobile communication systems, public exposure to electromagnetic fields (EMFs) raises the question of the impact of 900 MHz EMFs on cardiovascular health. Therefore, in this study, we experimentally investigated whether 915 MHz EMF exposure influenced cardiac metabolic, antioxidant, apoptotic, and fibro-inflammatory profiles in a mouse model. Healthy mice were sham-exposed or exposed to EMF for 14 days. Western blot analysis using whole cardiac tissue lysates demonstrated that there was no significant change in the expression of oxidative phosphorylation (OXPHOS) complexes between the control and EMF-exposed mice. In addition, the myocardial expression of fibro-inflammatory cytokines, antioxidant enzymes, and apoptosis-related markers remained unchanged in the EMF-challenged hearts. Finally, the structural integrity of the cardiac tissues was preserved among the groups. These findings suggest that the apoptotic, antioxidant, metabolic, and fibro-inflammatory profiles of the heart remained stable under conditions of EMF exposure in the analyzed mice.

Myocardial capacity of mitochondrial oxidative phosphorylation in response to prolonged electromagnetic stress.

Introduction: Mitochondria are central energy generators for the heart, producing adenosine triphosphate (ATP) through the oxidative phosphorylation (OXPHOS) system. However, mitochondria also guide critical cell decisions and responses to the environmental stressors. Methods: This study evaluated whether prolonged electromagnetic stress affects the mitochondrial OXPHOS system and structural modifications of the myocardium. To induce prolonged electromagnetic stress, mice were exposed to 915 MHz electromagnetic fields (EMFs) for 28 days. Results: Analysis of mitochondrial OXPHOS capacity in EMF-exposed mice pointed to a significant increase in cardiac protein expression of the Complex I, II, III and IV subunits, while expression level of α-subunit of ATP synthase (Complex V) was stable among groups. Furthermore, measurement of respiratory function in isolated cardiac mitochondria using the Seahorse XF24 analyzer demonstrated that prolonged electromagnetic stress modifies the mitochondrial respiratory capacity. However, the plasma level of malondialdehyde, an indicator of oxidative stress, and myocardial expression of mitochondria-resident antioxidant enzyme superoxide dismutase 2 remained unchanged in EMF-exposed mice as compared to controls. At the structural and functional state of left ventricles, no abnormalities were identified in the heart of mice subjected to electromagnetic stress. Discussion: Taken together, these data suggest that prolonged exposure to EMFs could affect mitochondrial oxidative metabolism through modulating cardiac OXPHOS system.

Interfacial S-O bonds specifically boost Z-scheme charge separation in a CuInS2/In2O3 heterojunction for efficient photocatalytic activity.

Reducing the recombination rate of photoexcited electron-hole pairs is always a great challenging work for the photocatalytic technique. In response to this issue, herein, a novel Z-scheme CuInS2/In2O3 with interfacial S-O linkages was synthesized by a hydrothermal and subsequently annealing method. The Fourier transform infrared (FT-IR) and X-ray photoelectron spectrometer (XPS) measurements confirmed the formation of covalent S-O bonds between CuInS2 and In2O3. The quenching and electron spin resonance (ESR) tests revealed the Z-scheme transfer route of photogenerated carriers over the CuInS2/In2O3 heterojunctions, which was further verified theoretically via density functional theory (DFT) calculations. As expected, the CuInS2/In2O3 heterojunctions showed significantly boosted photocatalytic activities for lomefloxacin degradation and Cr(vi) reduction under visible light illumination compared with the bare materials. Accordingly, a synergistic photocatalytic mechanism of Z-scheme heterostructures and interfacial S-O bonding was proposed, in which the S-O linkage could act as a specific bridge to modify the Z-scheme manner for accelerating the interfacial charge transmission. Furthermore, the CuInS2/In2O3 heterojunction also exhibited excellent performance perceived in the stability and reusability tests. This work provides a new approach for designing and fabricating novel Z-scheme heterostructures with a high-efficiency charge transfer route.

Three-dimensional modeling of microwave discharges in a waveguide-based plasma source with experimental comparison.

This paper proposes a transient three-dimensional model to simulate microwave-induced discharges in a waveguide-based plasma source under intermediate pressures. A plane-symmetric simplification method is applied to simplify half of the microwave plasma source in the calculation domain, dramatically reducing the demand for computational resources and calculation time. Meanwhile, the numerical simulations remain in three dimensions without dimensionality reduction, which allows us to directly calculate the efficiency of power coupling from the incident microwave to the plasma. Besides, the computation decrease improves the convergence performance of the mathematical model, making it possible to model the entire discharge process from 1×10^{-9} to 1×10^{4}s. This period covers the instantaneous microwave breakdown to the formation of a stable plasma column near steady state. The results have revealed the electromagnetic waveguide structure change of the microwave plasma source during the discharge process. Several microwave power-coupling efficiencies of the waveguide-based plasma source with different thicknesses and permittivities of the glass tube are calculated and compared with the experimentally measured data. Furthermore, the effects of the glass tube on the electromagnetic modes of the traveling microwave propagating along the plasma column and the discharge properties are also investigated. The numerically obtained results generally agree with the theoretical analysis and the experimental data in our previous studies, demonstrating the validity of the proposed mathematical model and the plane-symmetric simplification method.

Cardiac Cell Exposure to Electromagnetic Fields: Focus on Oxdative Stress and Apoptosis.

Exposure to electromagnetic fields (EMFs) is a sensitive research topic. Despite extensive research, to date there is no evidence to conclude that exposure to EMFs influences the cardiovascular system. In the present study, we examined whether 915 MHz EMF exposure affects myocardial antioxidative and apoptotic status in vitro and in vivo. No statistically significant difference in the apoptotic cell profile and antioxidant capacity was observed between controls and short-term EMF-exposed mouse cardiomyocytes and H9C2 cardiomyoblasts. Compared with sham-exposed controls, mice subjected to a 915 MHz EMF for 48 h and 72 h had no significant effect on structural tissue integrity and myocardial expression of apoptosis and antioxidant genes. Therefore, these results indicate that short-term exposure to EMF in cardiac cells and tissues did not translate into a significant effect on the myocardial antioxidant defense system and apoptotic cell death.