Asunto(s)
Infecciones Oportunistas Relacionadas con el SIDA/tratamiento farmacológico , Síndrome de Inmunodeficiencia Adquirida/complicaciones , Leishmaniasis Visceral/tratamiento farmacológico , Metronidazol/uso terapéutico , Tripanocidas/uso terapéutico , Adulto , Humanos , Leishmaniasis Visceral/complicaciones , Masculino , RecurrenciaAsunto(s)
Síndrome de Inmunodeficiencia Adquirida/complicaciones , Pasteurelosis Neumónica/etiología , Neumonía Bacteriana/etiología , Adulto , Líquido del Lavado Bronquioalveolar/microbiología , Humanos , Masculino , Pasteurella/aislamiento & purificación , Pasteurelosis Neumónica/tratamiento farmacológico , Pasteurelosis Neumónica/microbiología , Penicilina G/uso terapéutico , Penicilinas/uso terapéutico , Neumonía Bacteriana/tratamiento farmacológico , Neumonía Bacteriana/microbiología , Abuso de Sustancias por Vía Intravenosa/complicacionesRESUMEN
A 64-year-old man with alcoholic liver cirrhosis had a progressive decrease in the serum uric acid (UA) until it became undetectable, an increase renal UA clearance, mild glycosuria with normal serum glucose and a decrease in the tubular reabsorption of phosphate in association with cholestasis secondary to a gallbladder carcinoma. All these abnormalities returned to normal when the serum bilirubin levels decreased following surgical treatment. This clinical observation suggests that the reversible renal tubular transport defect was secondary to high levels of serum bilirubin.
Asunto(s)
Neoplasias de la Vesícula Biliar/fisiopatología , Hiperbilirrubinemia/sangre , Ácido Úrico/sangre , Bilirrubina/sangre , Glucemia/metabolismo , Colestasis/etiología , Estudios de Seguimiento , Neoplasias de la Vesícula Biliar/sangre , Neoplasias de la Vesícula Biliar/cirugía , Neoplasias de la Vesícula Biliar/orina , Glucosuria , Humanos , Hiperbilirrubinemia/complicaciones , Hiperbilirrubinemia/orina , Cirrosis Hepática Alcohólica/sangre , Cirrosis Hepática Alcohólica/complicaciones , Cirrosis Hepática Alcohólica/orina , Masculino , Persona de Mediana Edad , Factores de Tiempo , Ácido Úrico/orinaRESUMEN
The role of systemic lupus erythematosus (SLE) in the development of acute pancreatitis is a matter of controversy since, in many cases, this complication has been attributed to the drugs administered. In this study, we present a patient diagnosed as having SLE who developed acute pancreatitis with no apparent cause aside from her basic disease, and in whose necropsy was observed vascular damage consisting of severe intimal proliferation. Pancreatic vascular lesions in previously reported patients with lupus are reviewed.