The cost of being absent: Is meta-awareness of mind-wandering related to depression symptom severity, rumination tendencies and trauma intrusions?

BACKGROUND: Deng, Li and Tang (2014) reported that depression symptom severity is negatively associated with dispositional mindfulness and importantly, positively associated with zone-outs (mind-wandering without meta-awareness). We replicated and extended their study by exploring possible explanations for these relationships, and by also investigating whether mind-wandering is related to (1) trait rumination subtype-brooding, depressive or reflective, and (2) trauma intrusions-a hallmark PTSD symptom, since both rumination and trauma intrusions strongly correlate with depression. We also explored if dispositional mindfulness-the opposing construct of mind-wandering-mediated these relationships. METHOD: Two hundred participants completed mindfulness tendency and depression severity measures, counterbalanced with the Sustained Attention to Response Task (SART)-including thought probes to index behavioral mind-wandering (target-error frequency), subjective mind-wandering and meta-awareness-then the rumination style and trauma intrusion frequency measures. RESULTS: Depression scores positively correlated with mind-wandering with and without meta-awareness and with SART target-error rates, and negatively correlated with dispositional mindfulness. Further, trait brooding positively correlated with mind-wandering without meta-awareness. Dispositional mindfulness mediated the relationships between brooding and depression, and depression and mind-wandering, and also negatively correlated with trauma intrusion frequency. LIMITATIONS: Limitations include measurement and mind-wandering definitions, and an inability to make causal claims. CONCLUSIONS: People experiencing greater depression symptomology, and/or who have a greater tendency to brood, mind-wandered more often. Further, people who experience more trauma intrusions tend to be less mindful. These results point to potential harmful effects of mind-wandering through people's reduced propensity to be mindful, facilitating a negative self-referenced cognitive loop that may maintain or increase depression.

Why the COVID-19 pandemic is a traumatic stressor.

The COVID-19 pandemic does not fit into prevailing Post-traumatic Stress Disorder (PTSD) models, or diagnostic criteria, yet emerging research shows traumatic stress symptoms as a result of this ongoing global stressor. Current pathogenic event models focus on past, and largely direct, trauma exposure to certain kinds of life-threatening events. Yet, traumatic stress reactions to future, indirect trauma exposure, and non-Criterion A events exist, suggesting COVID-19 is also a traumatic stressor which could lead to PTSD symptomology. To examine this idea, we asked a sample of online participants (N = 1,040), in five western countries, to indicate the COVID-19 events they had been directly exposed to, events they anticipated would happen in the future, and other forms of indirect exposure such as through media coverage. We then asked participants to complete the Posttraumatic Stress Disorder Checklist-5, adapted to measure pre/peri/post-traumatic reactions in relation to COVID-19. We also measured general emotional reactions (e.g., angry, anxious, helpless), well-being, psychosocial functioning, and depression, anxiety, and stress symptoms. We found participants had PTSD-like symptoms for events that had not happened and when participants had been directly (e.g., contact with virus) or indirectly exposed to COVID-19 (e.g., via media). Moreover, 13.2% of our sample were likely PTSD-positive, despite types of COVID-19 "exposure" (e.g., lockdown) not fitting DSM-5 criteria. The emotional impact of "worst" experienced/anticipated events best predicted PTSD-like symptoms. Taken together, our findings support emerging research that COVID-19 can be understood as a traumatic stressor event capable of eliciting PTSD-like responses and exacerbating other related mental health problems (e.g., anxiety, depression, psychosocial functioning, etc.). Our findings add to existing literature supporting a pathogenic event memory model of traumatic stress.