[Tissue hypoxia in non-massive pulmonary arterial thromboembolism in patients with an implanted electrocardiostimulator].

The subjects of the study were 64 patients with coronary heart disease, atherosclerotic cardiosclerosis, constant atrial fibrillation, and total artificial atrioventricular blockade corrected by means of electrocardiostimulation, complicated by non-massive pulmonary arterial thromboembolism. Twenty-one patients had lowered arterial oxygen partial pressure, (paO2 whereas in 15 patients this variable was increased; both conditions were associated with vestibuloatactic and asthenic syndromes, as well as cognitive function decrease. The presence of sympathicotonia in 93% of the patients with increased paO2 made it possible to describe hyperventilation syndrome, which had not been described before and which takes place in cases of constantly relapsing non-massive embolization of pulmonary artery's small branches, associated with endocardial electrocardiostimulation.

[The role of opisthorchiasis in development of the secondary neurocirculatory dystonia and myocardial metabolic changes]. / Rol' opistorkhoza v razvitii vtorichnoi neirotsirkuliatornoi distonii i metabolicheskikh izmenenii miokarda.

AIM: Clarification of opisthorchiasis role in development of secondary neurocirculatory dystonia (NCD) and assessment of myocardial condition in patients with chronic opisthorchiasis (CO) with consideration of the disease duration and intensity of opisthorchotic invasion. MATERIAL AND METHODS: The trial enrolled 120 patients with CO, 35 patients with cardial NSD and 33 healthy controls. RESULTS: In long history of opisthorchiasis and severe invasion, autonomic cardiovascular regulation was abnormal. If CO patients have NCD, myocardial 99mTc-pyrophosphate scintigraphy reveals diffuse metabolic lesion of the myocardium. Long-term autonomic dysregulation of the cardiovascular system in CO and absence of adequate therapy lead to formation of progressive autonomic imbalance syndrome with sympathetic hypertonicity provoking metabolic disturbances of the myocardium.