RESUMEN
Echogenicity of the normal fetal telencephalic choroid plexus was characterized anatomically in 102 consecutive antenatal abdominal ultrasonographic examinations. Echogenicity of the plexus was observed only during the second trimester, first appearing at week 13 and disappearing after week 23. We suggest that this echogenicity may be due to different stages of glycogen chemistry in the plexus. The knowledge that telencephalic echogenicity is confined to the second trimester should help detect diseases that cause focal areas of echogenicity in fetal life.
Asunto(s)
Plexo Coroideo/diagnóstico por imagen , Plexo Coroideo/embriología , Ultrasonografía Prenatal , Adulto , Desarrollo Embrionario y Fetal , Femenino , Humanos , Embarazo , Segundo Trimestre del EmbarazoRESUMEN
Echogenicity of the telencephalic choroid plexus was observed in 34% of foetuses examined consecutively in routine obstetrical sonograms. Foetuses with echogenic were all aged between 13 and 23 weeks. 70% of the foetuses showing the echogenic plexus were in the second trimester. 80% of these foetuses were scanned during the fifth month of pregnancy (aged between 15 and 19 weeks). The biparietal diameter of 87% of the skulls with echogenic plexus ranged between 22 and 50mm. The characteristic appearance of the normal plexus during a certain period in the second trimester confirms its transient nature and suggests an underlying developmental mechanism. It appears and disappears well before the disease entities presenting in similar cephalic echogenicities.