Three Epstein-Barr virus latency proteins independently promote genomic instability by inducing DNA damage, inhibiting DNA repair and inactivating cell cycle checkpoints. | Oncogene;28(45): 3997-4008, 2009 Nov 12. | MEDLINE

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Three Epstein-Barr virus latency proteins independently promote genomic instability by inducing DNA damage, inhibiting DNA repair and inactivating cell cycle checkpoints.

Gruhne, B; Sompallae, R; Masucci, M G.

Affiliation

Gruhne B; Department of Cell and Molecular Biology, Karolinska Institutet, S-171 77 Stockholm, Sweden.

Oncogene ; 28(45): 3997-4008, 2009 Nov 12.

Article in En | MEDLINE | ID: mdl-19718051

Subject(s)

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: DNA Damage / Burkitt Lymphoma / Herpesvirus 4, Human / Genomic Instability / DNA Repair / Antigens, Viral Limits: Humans Language: En Journal: Oncogene Journal subject: BIOLOGIA MOLECULAR / NEOPLASIAS Year: 2009 Document type: Article Affiliation country:

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: DNA Damage / Burkitt Lymphoma / Herpesvirus 4, Human / Genomic Instability / DNA Repair / Antigens, Viral Limits: Humans Language: En Journal: Oncogene Journal subject: BIOLOGIA MOLECULAR / NEOPLASIAS Year: 2009 Document type: Article Affiliation country: