Studies on the defect which causes absence of decay accelerating factor (DAF) from the peripheral blood cells of an individual with the Inab phenotype. | Biochem J;261(2): 489-93, 1989 Jul 15. | MEDLINE

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Studies on the defect which causes absence of decay accelerating factor (DAF) from the peripheral blood cells of an individual with the Inab phenotype.

Tate, C G; Uchikawa, M; Tanner, M J; Judson, P A; Parsons, S F; Mallinson, G; Anstee, D J.

Affiliation

Tate CG; Department of Biochemistry, School of Medical Sciences, University of Bristol, U.K.

Biochem J ; 261(2): 489-93, 1989 Jul 15.

Article in En | MEDLINE | ID: mdl-2476116

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Blood Cells / Complement Inactivator Proteins / Blood Proteins / Membrane Proteins Type of study: Etiology_studies Limits: Humans Language: En Journal: Biochem J Year: 1989 Document type: Article Affiliation country:

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Blood Cells / Complement Inactivator Proteins / Blood Proteins / Membrane Proteins Type of study: Etiology_studies Limits: Humans Language: En Journal: Biochem J Year: 1989 Document type: Article Affiliation country: