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Opposing roles of p38 and JNK in a Drosophila model of TDP-43 proteinopathy reveal oxidative stress and innate immunity as pathogenic components of neurodegeneration.
Zhan, Lihong; Xie, Qijing; Tibbetts, Randal S.
Affiliation
  • Zhan L; Department of Human Oncology, Program in Molecular and Cellular Pharmacology, University of Wisconsin School of Medicine and Public Health, Madison, WI, USA.
  • Xie Q; Department of Human Oncology, Program in Molecular and Cellular Pharmacology, University of Wisconsin School of Medicine and Public Health, Madison, WI, USA.
  • Tibbetts RS; Department of Human Oncology, Program in Molecular and Cellular Pharmacology, University of Wisconsin School of Medicine and Public Health, Madison, WI, USA rstibbetts@wisc.edu.
Hum Mol Genet ; 24(3): 757-72, 2015 Feb 01.
Article in En | MEDLINE | ID: mdl-25281658
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Drosophila Proteins / JNK Mitogen-Activated Protein Kinases / Disease Models, Animal / DNA-Binding Proteins / Drosophila melanogaster / TDP-43 Proteinopathies Type of study: Prognostic_studies Limits: Animals / Humans Language: En Journal: Hum Mol Genet Journal subject: BIOLOGIA MOLECULAR / GENETICA MEDICA Year: 2015 Document type: Article Affiliation country:
Fulltext
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Drosophila Proteins / JNK Mitogen-Activated Protein Kinases / Disease Models, Animal / DNA-Binding Proteins / Drosophila melanogaster / TDP-43 Proteinopathies Type of study: Prognostic_studies Limits: Animals / Humans Language: En Journal: Hum Mol Genet Journal subject: BIOLOGIA MOLECULAR / GENETICA MEDICA Year: 2015 Document type: Article Affiliation country: