Altered p53 and NOX1 activity cause bioenergetic defects in a SCA7 polyglutamine disease model.
Biochim Biophys Acta
; 1847(4-5): 418-428, 2015.
Article
in En
| MEDLINE
| ID: mdl-25647692
Full text:
1
Collection:
01-internacional
Database:
MEDLINE
Main subject:
Peptides
/
Tumor Suppressor Protein p53
/
Spinocerebellar Ataxias
/
Disease Models, Animal
/
NADH, NADPH Oxidoreductases
/
Nerve Tissue Proteins
Type of study:
Prognostic_studies
Language:
En
Journal:
Biochim Biophys Acta
Year:
2015
Document type:
Article
Country of publication: