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Contribution of dopamine to mitochondrial complex I inhibition and dopaminergic deficits caused by methylenedioxymethamphetamine in mice.
Barros-Miñones, L; Goñi-Allo, B; Suquia, V; Beitia, G; Aguirre, N; Puerta, E.
Affiliation
  • Barros-Miñones L; Department of Pharmacology and Toxicology, School of Medicine, University of Navarra, Spain.
  • Goñi-Allo B; Department of Pharmacology and Toxicology, School of Medicine, University of Navarra, Spain.
  • Suquia V; Department of Pharmacology and Toxicology, School of Medicine, University of Navarra, Spain.
  • Beitia G; Department of Pharmacology and Toxicology, School of Medicine, University of Navarra, Spain.
  • Aguirre N; Department of Pharmacology and Toxicology, School of Medicine, University of Navarra, Spain.
  • Puerta E; Department of Pharmacology and Toxicology, School of Medicine, University of Navarra, Spain. Electronic address: epuerta@alumni.unav.es.
Neuropharmacology ; 93: 124-33, 2015 Jun.
Article in En | MEDLINE | ID: mdl-25666033
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Dopamine / N-Methyl-3,4-methylenedioxyamphetamine / Electron Transport Complex I / Hallucinogens Limits: Animals Language: En Journal: Neuropharmacology Year: 2015 Document type: Article Affiliation country:
Fulltext
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Dopamine / N-Methyl-3,4-methylenedioxyamphetamine / Electron Transport Complex I / Hallucinogens Limits: Animals Language: En Journal: Neuropharmacology Year: 2015 Document type: Article Affiliation country: