Your browser doesn't support javascript.
loading
Mice lacking neutral amino acid transporter B(0)AT1 (Slc6a19) have elevated levels of FGF21 and GLP-1 and improved glycaemic control.
Jiang, Yang; Rose, Adam J; Sijmonsma, Tjeerd P; Bröer, Angelika; Pfenninger, Anja; Herzig, Stephan; Schmoll, Dieter; Bröer, Stefan.
Affiliation
  • Jiang Y; Research School of Biology, The Australian National University, Canberra, ACT 0200, Australia.
  • Rose AJ; Joint Research Division Molecular Metabolic Control, German Cancer Research Center, Center for Molecular Biology, Heidelberg University and Heidelberg University Hospital, 69120 Heidelberg, Germany.
  • Sijmonsma TP; Joint Research Division Molecular Metabolic Control, German Cancer Research Center, Center for Molecular Biology, Heidelberg University and Heidelberg University Hospital, 69120 Heidelberg, Germany.
  • Bröer A; Research School of Biology, The Australian National University, Canberra, ACT 0200, Australia.
  • Pfenninger A; Sanofi-Aventis Deutschland GmbH, Industriepark Hoechst, Frankfurt am Main 65926, Germany.
  • Herzig S; Joint Research Division Molecular Metabolic Control, German Cancer Research Center, Center for Molecular Biology, Heidelberg University and Heidelberg University Hospital, 69120 Heidelberg, Germany.
  • Schmoll D; Sanofi-Aventis Deutschland GmbH, Industriepark Hoechst, Frankfurt am Main 65926, Germany.
  • Bröer S; Research School of Biology, The Australian National University, Canberra, ACT 0200, Australia.
Mol Metab ; 4(5): 406-17, 2015 May.
Article in En | MEDLINE | ID: mdl-25973388
Key words
Fulltext
Add to My VHL
Print
XML
PubMed Links
Search on Google

Full text: 1 Collection: 01-internacional Database: MEDLINE Type of study: Prognostic_studies Language: En Journal: Mol Metab Year: 2015 Document type: Article Affiliation country: Country of publication:
Fulltext
Add to My VHL
Print
XML
PubMed Links
Search on Google

Full text: 1 Collection: 01-internacional Database: MEDLINE Type of study: Prognostic_studies Language: En Journal: Mol Metab Year: 2015 Document type: Article Affiliation country: Country of publication: