Nitric oxide-mediated apoptosis in human breast cancer cells requires changes in mitochondrial functions and is independent of CD95 (APO-1/Fas). | Int J Oncol;16(1): 109-17, 2000 Jan. | MEDLINE

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Nitric oxide-mediated apoptosis in human breast cancer cells requires changes in mitochondrial functions and is independent of CD95 (APO-1/Fas).

Umansky, V; Ushmorov, A; Ratter, F; Chlichlia, K; Bucur, M; Lichtenauer, A; Rocha, M.

Affiliation

Umansky V; Deutsches Krebsforschungszentrum, Abteilung Zellulare Immunologie G0100, FSP Tumorimmunologie, D-69120 Heidelberg, Germany.

Int J Oncol ; 16(1): 109-17, 2000 Jan.

Article in En | MEDLINE | ID: mdl-10601555

Subject(s)

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Collection: 01-internacional Database: MEDLINE Main subject: Apoptosis / Fas Receptor / Mitochondria / Nitric Oxide Limits: Humans Language: En Journal: Int J Oncol Journal subject: NEOPLASIAS Year: 2000 Document type: Article Affiliation country: Alemania Country of publication: Grecia

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Collection: 01-internacional Database: MEDLINE Main subject: Apoptosis / Fas Receptor / Mitochondria / Nitric Oxide Limits: Humans Language: En Journal: Int J Oncol Journal subject: NEOPLASIAS Year: 2000 Document type: Article Affiliation country: Alemania Country of publication: Grecia