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Aromatic hydrocarbon receptor interaction with the retinoblastoma protein potentiates repression of E2F-dependent transcription and cell cycle arrest.
Puga, A; Barnes, S J; Dalton, T P; Chang, C y; Knudsen, E S; Maier, M A.
Affiliation
  • Puga A; Center for Environmental Genetics, Department of Environmental Health, University of Cincinnati Medical Center, Cincinnati, Ohio 45267-0056, USA. Alvaro.Puga@UC.EDU
J Biol Chem ; 275(4): 2943-50, 2000 Jan 28.
Article in En | MEDLINE | ID: mdl-10644764
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Collection: 01-internacional Database: MEDLINE Main subject: Transcription Factors / Transcription, Genetic / Carrier Proteins / Retinoblastoma Protein / Receptors, Aryl Hydrocarbon / Cell Cycle Proteins / DNA-Binding Proteins Limits: Animals / Humans Language: En Journal: J Biol Chem Year: 2000 Document type: Article Affiliation country: Estados Unidos Country of publication: Estados Unidos
Search on Google
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Collection: 01-internacional Database: MEDLINE Main subject: Transcription Factors / Transcription, Genetic / Carrier Proteins / Retinoblastoma Protein / Receptors, Aryl Hydrocarbon / Cell Cycle Proteins / DNA-Binding Proteins Limits: Animals / Humans Language: En Journal: J Biol Chem Year: 2000 Document type: Article Affiliation country: Estados Unidos Country of publication: Estados Unidos