Activation of invariant NKT cells ameliorates experimental ocular autoimmunity by a mechanism involving innate IFN-gamma production and dampening of the adaptive Th1 and Th17 responses.

Grajewski, Rafael S; Hansen, Anna M; Agarwal, Rajeev K; Kronenberg, Mitchell; Sidobre, Stephane; Su, Shao Bo; Silver, Phyllis B; Tsuji, Moriya; Franck, Richard W; Lawton, Anne P; Chan, Chi-Chao; Caspi, Rachel R

Grajewski, Rafael S; Hansen, Anna M; Agarwal, Rajeev K; Kronenberg, Mitchell; Sidobre, Stephane; Su, Shao Bo; Silver, Phyllis B; Tsuji, Moriya; Franck, Richard W; Lawton, Anne P; Chan, Chi-Chao; Caspi, Rachel R.

Affiliation

Grajewski RS; Laboratory of Immunology, National Eye Institute, National Institutes of Health, Bethesda, MD 20892, USA.

J Immunol ; 181(7): 4791-7, 2008 Oct 01.

Article in En | MEDLINE | ID: mdl-18802082

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Autoimmune Diseases / Uveitis / Lymphocyte Activation / Interferon-gamma / Th1 Cells / Interleukin-17 / Natural Killer T-Cells Type of study: Prognostic_studies Limits: Animals Language: En Journal: J Immunol Year: 2008 Document type: Article Affiliation country: Estados Unidos Country of publication: Estados Unidos

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