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BCR-ABL suppresses autophagy through ATF5-mediated regulation of mTOR transcription.
Sheng, Zhi; Ma, Leyuan; Sun, Jiaoyuan E; Zhu, Lihua J; Green, Michael R.
Affiliation
  • Sheng Z; Howard Hughes Medical Institute, Program in Gene Function and Expression, University of Massachusetts Medical School, Worcester, MA 01605, USA.
Blood ; 118(10): 2840-8, 2011 Sep 08.
Article in En | MEDLINE | ID: mdl-21715304
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Autophagy / Leukemia, Myelogenous, Chronic, BCR-ABL Positive / Gene Expression Regulation, Neoplastic / Fusion Proteins, bcr-abl / Activating Transcription Factors / TOR Serine-Threonine Kinases Type of study: Prognostic_studies Limits: Animals / Humans Language: En Journal: Blood Year: 2011 Document type: Article Affiliation country: Estados Unidos Country of publication: Estados Unidos
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Autophagy / Leukemia, Myelogenous, Chronic, BCR-ABL Positive / Gene Expression Regulation, Neoplastic / Fusion Proteins, bcr-abl / Activating Transcription Factors / TOR Serine-Threonine Kinases Type of study: Prognostic_studies Limits: Animals / Humans Language: En Journal: Blood Year: 2011 Document type: Article Affiliation country: Estados Unidos Country of publication: Estados Unidos