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Proline, glutamic acid and leucine-rich protein-1 is essential for optimal p53-mediated DNA damage response.
Nair, B C; Krishnan, S R; Sareddy, G R; Mann, M; Xu, B; Natarajan, M; Hasty, P; Brann, D; Tekmal, R R; Vadlamudi, R K.
Affiliation
  • Nair BC; University of Texas Health Science Center, and Cancer Therapy and Research Center, San Antonio, TX, USA.
  • Krishnan SR; University of Texas Health Science Center, and Cancer Therapy and Research Center, San Antonio, TX, USA.
  • Sareddy GR; University of Texas Health Science Center, and Cancer Therapy and Research Center, San Antonio, TX, USA.
  • Mann M; University of Texas Health Science Center, and Cancer Therapy and Research Center, San Antonio, TX, USA.
  • Xu B; Molecular Radiation Biology Laboratory, Research Institute, South Birmingham, AL, USA.
  • Natarajan M; University of Texas Health Science Center, and Cancer Therapy and Research Center, San Antonio, TX, USA.
  • Hasty P; University of Texas Health Science Center, and Cancer Therapy and Research Center, San Antonio, TX, USA.
  • Brann D; Institute of Molecular Medicine and Genetics, Georgia Reagents University, Augusta, GA, USA.
  • Tekmal RR; University of Texas Health Science Center, and Cancer Therapy and Research Center, San Antonio, TX, USA.
  • Vadlamudi RK; University of Texas Health Science Center, and Cancer Therapy and Research Center, San Antonio, TX, USA.
Cell Death Differ ; 21(9): 1409-18, 2014 Sep.
Article in En | MEDLINE | ID: mdl-24786831

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Transcription Factors / DNA Damage / Tumor Suppressor Protein p53 / Co-Repressor Proteins Limits: Humans Language: En Journal: Cell Death Differ Year: 2014 Document type: Article Affiliation country: Estados Unidos Country of publication: Reino Unido

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Transcription Factors / DNA Damage / Tumor Suppressor Protein p53 / Co-Repressor Proteins Limits: Humans Language: En Journal: Cell Death Differ Year: 2014 Document type: Article Affiliation country: Estados Unidos Country of publication: Reino Unido