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Muscle-specific CRISPR/Cas9 dystrophin gene editing ameliorates pathophysiology in a mouse model for Duchenne muscular dystrophy.
Bengtsson, Niclas E; Hall, John K; Odom, Guy L; Phelps, Michael P; Andrus, Colin R; Hawkins, R David; Hauschka, Stephen D; Chamberlain, Joel R; Chamberlain, Jeffrey S.
Affiliation
  • Bengtsson NE; Department of Neurology, University of Washington, Seattle, Washington 98195-7720, USA.
  • Hall JK; Senator Paul D. Wellstone Muscular Dystrophy Cooperative Research Center, University of Washington, Seattle, Washington 98195-7720, USA.
  • Odom GL; Department of Neurology, University of Washington, Seattle, Washington 98195-7720, USA.
  • Phelps MP; Senator Paul D. Wellstone Muscular Dystrophy Cooperative Research Center, University of Washington, Seattle, Washington 98195-7720, USA.
  • Andrus CR; Department of Neurology, University of Washington, Seattle, Washington 98195-7720, USA.
  • Hawkins RD; Senator Paul D. Wellstone Muscular Dystrophy Cooperative Research Center, University of Washington, Seattle, Washington 98195-7720, USA.
  • Hauschka SD; Department of Pathology, University of Washington, Seattle, Washington 98195-7720, USA.
  • Chamberlain JR; Department of Medicine, University of Washington, Seattle, Washington 98195-7720, USA.
  • Chamberlain JS; Department of Genome Sciences, University of Washington, Seattle, Washington 98195-7720, USA.
Nat Commun ; 8: 14454, 2017 02 14.
Article in En | MEDLINE | ID: mdl-28195574

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Dystrophin / Muscular Dystrophy, Duchenne / CRISPR-Cas Systems / Gene Editing Limits: Animals Language: En Journal: Nat Commun Journal subject: BIOLOGIA / CIENCIA Year: 2017 Document type: Article Affiliation country: Estados Unidos Country of publication: Reino Unido

Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Dystrophin / Muscular Dystrophy, Duchenne / CRISPR-Cas Systems / Gene Editing Limits: Animals Language: En Journal: Nat Commun Journal subject: BIOLOGIA / CIENCIA Year: 2017 Document type: Article Affiliation country: Estados Unidos Country of publication: Reino Unido