Amyloid-ß oligomers transiently inhibit AMP-activated kinase and cause metabolic defects in hippocampal neurons.
J Biol Chem
; 292(18): 7395-7406, 2017 05 05.
Article
in En
| MEDLINE
| ID: mdl-28302722
Key words
Full text:
1
Collection:
01-internacional
Database:
MEDLINE
Main subject:
Peptide Fragments
/
Amyloid beta-Peptides
/
Amyloid beta-Protein Precursor
/
AMP-Activated Protein Kinases
/
Alzheimer Disease
/
Hippocampus
/
Neurons
Limits:
Animals
/
Humans
Language:
En
Journal:
J Biol Chem
Year:
2017
Document type:
Article
Country of publication:
Estados Unidos