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Disruption of the mitochondria-associated ER membrane (MAM) plays a central role in palmitic acid-induced insulin resistance.
Shinjo, Satoko; Jiang, Shuying; Nameta, Masaaki; Suzuki, Tomohiro; Kanai, Mai; Nomura, Yuta; Goda, Nobuhito.
Affiliation
  • Shinjo S; Department of Life Science and Medical Bioscience, School of Advanced Science and Engineering, Waseda University, Tokyo 162-8480, Japan; Research Institute for Life Support Innovation, Research Organization for Nano & Life Innovation, Waseda University, Tokyo 162-0041, Japan. Electronic address:
  • Jiang S; Niigata College of Medical Technology, Niigata 950-2076, Japan.
  • Nameta M; Department of Electron Microscope Core Facility, Niigata University, Niigata 951-8510, Japan.
  • Suzuki T; Department of Life Science and Medical Bioscience, School of Advanced Science and Engineering, Waseda University, Tokyo 162-8480, Japan.
  • Kanai M; Department of Life Science and Medical Bioscience, School of Advanced Science and Engineering, Waseda University, Tokyo 162-8480, Japan; School of Creative Science and Engineering, Waseda University, Tokyo 162-8480, Japan.
  • Nomura Y; Department of Life Science and Medical Bioscience, School of Advanced Science and Engineering, Waseda University, Tokyo 162-8480, Japan.
  • Goda N; Department of Life Science and Medical Bioscience, School of Advanced Science and Engineering, Waseda University, Tokyo 162-8480, Japan.
Exp Cell Res ; 359(1): 86-93, 2017 10 01.
Article in En | MEDLINE | ID: mdl-28827061
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Insulin Resistance / Palmitic Acid / Endoplasmic Reticulum / Intracellular Membranes / Mitochondria Type of study: Risk_factors_studies Limits: Humans Language: En Journal: Exp Cell Res Year: 2017 Document type: Article Country of publication: Estados Unidos
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Insulin Resistance / Palmitic Acid / Endoplasmic Reticulum / Intracellular Membranes / Mitochondria Type of study: Risk_factors_studies Limits: Humans Language: En Journal: Exp Cell Res Year: 2017 Document type: Article Country of publication: Estados Unidos