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Disruption of glucagon receptor signaling causes hyperaminoacidemia exposing a possible liver-alpha-cell axis.
Galsgaard, Katrine D; Winther-Sørensen, Marie; Ørskov, Cathrine; Kissow, Hannelouise; Poulsen, Steen S; Vilstrup, Hendrik; Prehn, Cornelia; Adamski, Jerzy; Jepsen, Sara L; Hartmann, Bolette; Hunt, Jenna; Charron, Maureen J; Pedersen, Jens; Wewer Albrechtsen, Nicolai J; Holst, Jens J.
Affiliation
  • Galsgaard KD; Department of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen , Copenhagen , Denmark.
  • Winther-Sørensen M; Novo Nordisk Foundation Center for Basic Metabolic Research, Faculty of Health and Medical Sciences, University of Copenhagen , Copenhagen , Denmark.
  • Ørskov C; Department of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen , Copenhagen , Denmark.
  • Kissow H; Novo Nordisk Foundation Center for Basic Metabolic Research, Faculty of Health and Medical Sciences, University of Copenhagen , Copenhagen , Denmark.
  • Poulsen SS; Department of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen , Copenhagen , Denmark.
  • Vilstrup H; Department of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen , Copenhagen , Denmark.
  • Prehn C; Novo Nordisk Foundation Center for Basic Metabolic Research, Faculty of Health and Medical Sciences, University of Copenhagen , Copenhagen , Denmark.
  • Adamski J; Department of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen , Copenhagen , Denmark.
  • Jepsen SL; Department of Hepato-Gastroenterology, Aarhus University Hospital , Aarhus , Denmark.
  • Hartmann B; Institute of Experimental Genetics, Genome Analysis Center, Helmholtz Zentrum, German Research Center for Environmental Health, München-Neuerberg, Germany.
  • Hunt J; Institute of Experimental Genetics, Genome Analysis Center, Helmholtz Zentrum, German Research Center for Environmental Health, München-Neuerberg, Germany.
  • Charron MJ; Lehrstul für Experimentelle Genetik, Technishe Universität München, Freising- Weihenstephan , Germany.
  • Pedersen J; German Center for Diabetes Research, München-Nueherberg, Germany.
  • Wewer Albrechtsen NJ; Department of Biomedical Sciences, Faculty of Health and Medical Sciences, University of Copenhagen , Copenhagen , Denmark.
  • Holst JJ; Novo Nordisk Foundation Center for Basic Metabolic Research, Faculty of Health and Medical Sciences, University of Copenhagen , Copenhagen , Denmark.
Am J Physiol Endocrinol Metab ; 314(1): E93-E103, 2018 01 01.
Article in En | MEDLINE | ID: mdl-28978545
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Cell Communication / Receptors, Glucagon / Hepatocytes / Glucagon-Secreting Cells / Amino Acids Type of study: Etiology_studies Limits: Animals Language: En Journal: Am J Physiol Endocrinol Metab Journal subject: ENDOCRINOLOGIA / FISIOLOGIA / METABOLISMO Year: 2018 Document type: Article Affiliation country: Dinamarca Country of publication: Estados Unidos
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Cell Communication / Receptors, Glucagon / Hepatocytes / Glucagon-Secreting Cells / Amino Acids Type of study: Etiology_studies Limits: Animals Language: En Journal: Am J Physiol Endocrinol Metab Journal subject: ENDOCRINOLOGIA / FISIOLOGIA / METABOLISMO Year: 2018 Document type: Article Affiliation country: Dinamarca Country of publication: Estados Unidos