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Altered ER-mitochondria contact impacts mitochondria calcium homeostasis and contributes to neurodegeneration in vivo in disease models.
Lee, Kyu-Sun; Huh, Sungun; Lee, Seongsoo; Wu, Zhihao; Kim, Ae-Kyeong; Kang, Ha-Young; Lu, Bingwei.
Affiliation
  • Lee KS; Department of Pathology, Stanford University School of Medicine, Stanford, CA 94305.
  • Huh S; Program of Neuroscience, Stanford University School of Medicine, Stanford, CA 94305.
  • Lee S; Program of Cancer Biology, Stanford University School of Medicine, Stanford, CA 94305.
  • Wu Z; BioNanotechnology Research Center, Korea Research Institute of Biotechnology and Bioscience, 34141 Daejeon, Korea.
  • Kim AK; Department of Pathology, Stanford University School of Medicine, Stanford, CA 94305.
  • Kang HY; Program of Neuroscience, Stanford University School of Medicine, Stanford, CA 94305.
  • Lu B; Program of Cancer Biology, Stanford University School of Medicine, Stanford, CA 94305.
Proc Natl Acad Sci U S A ; 115(38): E8844-E8853, 2018 09 18.
Article in En | MEDLINE | ID: mdl-30185553
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Parkinson Disease / Calcium / Drosophila melanogaster / Endoplasmic Reticulum / Mitochondria Type of study: Prognostic_studies Limits: Animals / Humans Language: En Journal: Proc Natl Acad Sci U S A Year: 2018 Document type: Article Country of publication: Estados Unidos
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Parkinson Disease / Calcium / Drosophila melanogaster / Endoplasmic Reticulum / Mitochondria Type of study: Prognostic_studies Limits: Animals / Humans Language: En Journal: Proc Natl Acad Sci U S A Year: 2018 Document type: Article Country of publication: Estados Unidos