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α-synuclein strains that cause distinct pathologies differentially inhibit proteasome.
Suzuki, Genjiro; Imura, Sei; Hosokawa, Masato; Katsumata, Ryu; Nonaka, Takashi; Hisanaga, Shin-Ichi; Saeki, Yasushi; Hasegawa, Masato.
Affiliation
  • Suzuki G; Dementia Research Project, Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan.
  • Imura S; Dementia Research Project, Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan.
  • Hosokawa M; Laboratory of Molecular Neuroscience, Department of Biological Sciences, Tokyo Metropolitan University, Tokyo, Japan.
  • Katsumata R; Dementia Research Project, Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan.
  • Nonaka T; Dementia Research Project, Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan.
  • Hisanaga SI; Dementia Research Project, Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan.
  • Saeki Y; Laboratory of Molecular Neuroscience, Department of Biological Sciences, Tokyo Metropolitan University, Tokyo, Japan.
  • Hasegawa M; Protein Metabolism Project, Tokyo Metropolitan Institute of Medical Science, Tokyo, Japan.
Elife ; 92020 07 22.
Article in En | MEDLINE | ID: mdl-32697196
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Proteasome Endopeptidase Complex / Alpha-Synuclein / Neurons Limits: Animals Language: En Journal: Elife Year: 2020 Document type: Article Affiliation country: Japón Country of publication: Reino Unido
Fulltext
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Proteasome Endopeptidase Complex / Alpha-Synuclein / Neurons Limits: Animals Language: En Journal: Elife Year: 2020 Document type: Article Affiliation country: Japón Country of publication: Reino Unido