Deficiency in the function of inhibitory interneurons contributes to glutamate-associated central sensitization through GABABR2-SynCAM1 signaling in chronic migraine rats.

Zeng, Xiaoxu; Niu, Yingying; Qin, Guangcheng; Zhang, Dunke; Zhou, Jiying; Chen, Lixue

Zeng, Xiaoxu; Niu, Yingying; Qin, Guangcheng; Zhang, Dunke; Zhou, Jiying; Chen, Lixue.

Affiliation

Zeng X; Laboratory Research Center, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China.
Niu Y; Laboratory Research Center, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China.
Qin G; Laboratory Research Center, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China.
Zhang D; Laboratory Research Center, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China.
Zhou J; Department of Neurology, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China.
Chen L; Laboratory Research Center, The First Affiliated Hospital of Chongqing Medical University, Chongqing, China.

FASEB J ; 34(11): 14780-14798, 2020 11.

Article in En | MEDLINE | ID: mdl-32931071

Subject(s)
Key words

CGRP; PAG; PKA; c-Fos; glutamate

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Immunoglobulins / Signal Transduction / Cell Adhesion Molecules / Receptors, GABA-B / Central Nervous System Sensitization / Interneurons / Migraine Disorders Type of study: Risk_factors_studies Limits: Animals Language: En Journal: FASEB J Journal subject: BIOLOGIA / FISIOLOGIA Year: 2020 Document type: Article Affiliation country: China Country of publication: Estados Unidos

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